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12篇 您的检索式:作者名="Peverini"
    题名 作者 年代 出处 被引量
1Obstructive Sleep Apnea Syndrome: Blood Viscosity, Blood Coagulation Abnormalities, and Early Atherosclerosis显示文摘Domenico Maurizio Toraldo Francesco Peverini Michele Benedetto Francesco Nuccio 2013Lung2013,,1:2
2QTC interval in infants receiving cisapvied 显示文摘Chhina S Peverini RL Deminq DD 2002J Perinato12002,22,1:1
3Obstructive sleep apnea syndrome: blood viscosity, blood coagulation abnormalities, and early atherosclerosls显示文摘Toraldo DM Peverini F De Benedetto M 2013Lung2013,191,:1
4Obstructive sleep apnea syndrome : blood viscosity, blood coagulation abnormalities, and early atherosclerosis 显示文摘Toraldo DM Peverini F De Benedetto M 2013Lung2013,191,:1
5QTC interval in infants receiving cisapvied 显示文摘Chhina S Peverini RL Deminq DD 2002J Perinatol2002,22,1:1
6Ion beam profiling of aspherical X-ray mirrors显示文摘PEVERINI L KOZHEVNIKOV I ROMMEVEAUX A 2010Nuclear Instruments and Methods in Physics Research Section A: Accelerators Spectrometers Detectors and Associated Equipment2010,616,23:1
7Severe oxygen desaturation in obstructive sleep-apnea syndrome as a possible cause of ischemic stroke显示文摘Sacco S Peverini F Bastianello S 2005Eur Neurol2005,53,2:1
8Obstructive sleep apnea syndrome: blood viscosity, blood coagulation abnormalities, and early atherosclerosis 显示文摘Toraldo DM Peverini F De Benedetto M 2013Lung2013,191,:1
9Obstructive sleep apnea syndrome: blood viscosity, blood coagulation abnormalities,and early atherosclerosis显示文摘Toraldo DM Peverini F De Bencdetto M 2013Lung2013,191,1:1
10Obstructive sleep apnea syndrome:blood viscosity,blood coagu- lation abnormalities,and early atherosclerosis显示文摘TORALDO DMl PEVERINI F DE BENEDETTO M 2013Lung2013,191,1:1
11Roughness Evolution of Si Surfaces Upon Ion Erosion 显示文摘DE ROOIJ-LOHMANN V I T A KOZHEVNIKOV I V PEVERINI L 2010Applied Surface Science2010,256,:1
12Changes in the redox status of the brain in an experimental glaucoma model显示文摘The purpose of this study was to evaluate the redox status changes of primary visual targets in the rat brain of a high pressure-induced glaucoma model.The animal model consisted of inducing ocular hypertension by cauterizing two episcleral veins on the left eye.The markers of oxidative damage and the oxidative balance evaluated in the brain seven days postoperative were:nitrites concentration,levels of non-enzymatic antioxidants and antiox-idant enzymes activity.The increase in the nitrite content,which could be the result of the enhancement in the production of nitrogen species,and in the activity of NADPH oxidase in the glaucoma group could lead to an increase on lipid and protein damage.The decrease on the non-enzymatic antioxidants and the compensatory increase of the superoxide dismutase and glutathione peroxidase activities could be a consequence of the increase of oxidative processes.The decrease in the activity of glutathione reductase leads to a decrease in the recycling of thiol groups.We suggest that oxidative stress can possibly acts as a risk factor for neurodegeneration in the brain.Therapeu-tic strategies to stop the progression of the disease in glaucoma should also be considered the central neuronal degeneration beyond the retina and the optic nerve.CLAUDIA G.REIDES ROMINA M.LASAGNI VITAR AGUSTINA PEVERINI NATASHA S.JANEZIC AILEN G.HVOZDA ARANA SANDRA M.FERREIRA SUSANA F.LLESUY 2016BIOCELL2016,40,1:0
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