| 1 | Psychosocial mechanisms for the transmission of somatic symptoms from parents to children显示文摘AIM: To examine familial aggregation of irritable bowel syndrome(IBS) via parental reinforcement/modeling of symptoms, coping, psychological distress, and exposure to stress.METHODS:Mothers of children between the ages of8 and 15 years with and without IBS were identified through the Group Health Cooperative of Puget Sound.Mothers completed questionnaires,including the Child Behavior Checklist(child psychological distress),the Family Inventory of Life Events(family exposure to stress),SCL-90R(mother psychological distress),and the Pain Response Inventory(beliefs about pain).Children were interviewed separately from their parents and completed the Pain Beliefs Questionnaire(beliefs about pain),Pain Response Inventory(coping)and Child Symptom Checklist[gastrointestinal(GI)symptoms].In addition,health care utilization data was obtained from the automated database of Group Health Cooperative.Mothers with IBS(n=207)and their 296 children were compared to 240 control mothers and their 335 children,while controlling for age and education.RESULTS:Hypothesis 1:reinforcement of expression of GI problems is only related to GI symptoms,but not others(cold symptoms)in children.There was no significant correlation between parental reinforcement of symptoms and child expression of GI or other symptoms.Hypothesis 2:modeling of GI symptomsis related to GI but not non-GI symptom reporting in children.Children of parents with IBS reported more non-GI(8.97 vs 6.70,P<0.01)as well as more GI(3.24 vs 2.27,P<0.01)symptoms.Total health care visits made by the mother correlated with visits made by the child(rho=0.35,P<0.001 for cases,rho=0.26,P<0.001 for controls).Hypothesis 3:children learn to share the methods of coping with illness that their mothers exhibit.Methods used by children to cope with stomachaches differed from methods used by their mothers.Only 2/16 scales showed weak but significant correlations(stoicism rho=0.13,P<0.05;acceptance rho=0.13,P<0.05).Hypothesis 4:mothers and children share psychological traits such as anxiety,depression,and somatization.Child psychological distress correlated with mother’s psychological distress(rho=0.41,P<0.001 for cases,rho=0.38,P<0.001 for controls).Hypothesis 5:stress that affects the whole family might explain the similarities between mothers and their children.Family exposure to stress was not a significant predictor of children’s symptom reports.Hypothesis 6:the intergenerational transmission of GI illness behavior may be due to multiple mechanisms.Regression analysis identified multiple independent predictors of the child’s GI complaints,which were similar to the predictors of the child’s non-GI symptoms(mother’s IBS status,child psychological symptoms,child catastrophizing,and child age).CONCLUSION:Multiple factors influence the reporting of children’s gastrointestinal and non-gastrointestinal symptoms.The clustering of illness within families is best understood using a model that incorporates all these factors. | Miranda AL van Tilburg Rona L Levy Lynn S Walker Michael Von Korff Lauren D Feld Michelle Garner Andrew D Feld William E Whitehead | 2015 | World Journal of Gastroenterology2015,21,18: | 2 |
| 2 | Measuring and Visualizing Research Collaboration and Productivity显示文摘Purpose: This paper presents findings of a quasi-experimental assessment to gauge the research productivity and degree of interdisciplinarity of research center outputs. Of special interest, we share an enriched visualization of research co-authoring patterns. Design/methodology/approach: We compile publications by 45 researchers in each of 1) the i UTAH project, which we consider here to be analogous to a 'research center,' 2) CG1— a comparison group of participants in two other Utah environmental research centers, and 3) CG2—a comparison group of Utah university environmental researchers not associated with a research center. We draw bibliometric data from Web of Science and from Google Scholar. We gather publications for a period before i UTAH had been established(2010–2012) and a period after(2014–2016). We compare these research outputs in terms of publications and citations thereto. We also measure interdisciplinarity using Integration scoring and generate science overlay maps to locate the research publications across disciplines.Findings: We find that participation in the i UTAH project appears to increase research outputs(publications in the After period) and increase research citation rates relative to the comparison group researchers(although CG1 research remains most cited, as it was in the Before period). Most notably, participation in i UTAH markedly increases co-authoring among researchers—in general; and for junior, as well as senior, faculty; for men and women: across organizations; and across disciplines.Research limitations: The quasi-experimental design necessarily generates suggestive, not definitively causal, findings because of the imperfect controls.Practical implications: This study demonstrates a viable approach for research assessment of a center or program for which random assignment of control groups is not possible. It illustrates use of bibliometric indicators to inform R&D program management.Originality/value: New visualizations of researcher collaboration provide compelling comparisons of the extent and nature of social networking among target cohorts. | Jon Garner Alan L.Porter Andreas Leidolf Michelle Baker | 2018 | Journal of Data and Information Science2018,3,1: | 0 |