|
|
|
题名
|
作者
|
年代
|
出处
|
被引量
|
| 1 | Differential roles of hydrogen peroxide and superoxide in mediating IL-1-induced NF-kappaB activation and iNOS expression in bovine articular chondrocytes显示文摘 | Mendes AF Caramona MM Carvalho AP | 2003 | J Cell Biochem2003，88，4： | 1 |
| 2 | Differential roles of hydrogen peroxide and superoxide in mediating IL-1-induced NF-kappaB activation and iNOS expression in bovine articular chondrocytes显示文摘 | Mendes AF Caramona MM Carvalho AP | 2003 | J Cell Biochem2003，88，4： | 1 |
| 3 | Brain activation of SIRT1 : role in neuropathology 显示文摘 | Parafso AF Mendes KL Santos SH | 2013 | Mol Neurobiol2013，48，3： | 1 |
| 4 | Differential roles of hydrogen peroxide and superoxide in mediating IL-1 induced NF-kappaB activation and iNOS expression in bovine articular chondrocytes 显示文摘 | Mendes AF Caramona MM Carvalho AP | 2003 | J Cell Biochem2003，88，4： | 1 |
| 5 | Diphenyleneiodonium inhabits NF-κB activation and Inos expression induced by IL-1 beta:involvement of reactive oxygen species显示文摘 | Mendes AF Carvalho AP Caramona MM | 2001 | Mediators Inflamm2001，10，： | 1 |
| 6 | Use of high-poweredmagnification to detect occlusal caries in primary teeth显示文摘 | Mendes FM Ganzerla E Nunes AF | 2006 | Am JDent2006，19，1： | 1 |
| 7 | Differential roles of hydrogen peroxide and superoxide in mediating IL-1 induced NF-kappaB activation and iNOS expression in bovine articular chondrocytes显示文摘 | Mendes AF Caramona MM Carvalho AP et aI | | 0，，04： | 1 |
| 8 | Differential roles of hydrogen peroxide and superoxide in mediating IL-1-induced NF-kappa B activation and iNOS expression in bovine articular chondrocytes显示文摘 | Mendes AF Caramona MM Carvalho AP | 2003 | Cell Biochem2003，88，4： | 1 |
| 9 | Hydrogen peroxide mediates interleukin - 1 beta - induced AP - 1 activation in articular chondrocytes: implications for the regulation of iNOS expression 显示文摘 | Mendes AF Caramona MM Carvalho AP | 2003 | Cell Biol Toxicol2003，19，4： | 1 |
| 10 | Differential roles of hydrogen peroxide and superoxide in mediating IL-1 inducedNF-kappaB activation and iNOS expression in bovine articular chondrocytes显示文摘 | Mendes AF Caramona MM Carvalho AP | 2003 | J Cell Biochem2003，88，4： | 1 |
| 11 | Differen- tial roles of hydrogen peroxide and superoxide in media- ting IL-1 induced NF-kappB aetivation and iNOS expres- sion in bovine artieular chondrocytes显示文摘 | Mendes AF Caramona MM Carvalho AP | 2003 | Cell Biochem2003，88，4： | 1 |
| 12 | Role of mitogen-activated protein kinases and tyrosine kinases on IL-1-induced NF-κB activation and iNOS expression in bovine articular chondrocytes显示文摘 | Mendes AF Caramona MM Carvalho AP | 2002 | Nitric Oxide2002，6，1： | 1 |
| 13 | Diacerhein and rhein prevent interleukin-1 beta induced nuclear factor-kappa B activation by inhibiting the degradation of inhibitor kappa B-alpha显示文摘 | Mendes AF Caramona MM Carvalho AP | 2002 | Pharmacol Toxicol2002，91，： | 1 |
| 14 | Supercritical CO2 extraction of γ-linolenic acid and other lipids from Arthrospira(Spirulina)maxima:Comparison with organic solvent extraction显示文摘 | Mendes RL Reis AD Palavra AF | | 0，，01： | 1 |
| 15 | Diacerhein and rhein prevent interleukin-1 beta induced nuclear factor kappa B activation by inhibiting the degradation of inhibitor kappa B Alpha 显示文摘 | Mendes AF Caramona MM Carvalho AP | 2002 | Pharmacol Toxicol2002，91，： | 1 |
| 16 | Supercritical CO2 extraction of γ- linolenie acid (GLA) and other lipids from Arthrospira(Spirulina) maxima : Comparison with organic solvent extraction 显示文摘 | Mendes RL Reis AD Palavra AF | 2006 | Food Chem2006，99，1： | 1 |
| 17 | Diacerhein and rhein prevent interleukin-1 beta-induced nuclear factor-kappaB activation by inhibiting the degradation of inhibitor kappa B-alpha显示文摘 | Mendes AF Caramona MM de Carvalho AP | | 0，，： | 1 |
| 18 | Diacerhein and rhein prevent interleukin-1beta-induced nuclear factor-kappaB activation by inhibiting the degradation of inhibitor kappaB-alpha显示文摘 | Caramona MM de Carvalho AP | 2002 | Pharmacology & Toxicology2002，91，1： | 1 |
| 19 | Prognostic factors for progression of liver structural lesions in chronic hepatitis C patients显示文摘AIM: To evaluate the epidemiological, clinical, laboratory and histological variables capable of predicting the progression of hepatic structural disturbances in chronic hepatitis C patients during the time interval between two liver biopsies. METHODS: Clinical charts of 112 chronic hepatitis C patients were retrospectively analyzed, whereas liver biopsies were revised. Immunohistochemical detection of interferon receptor was based on the Envision-Peroxidase System. RESULTS: In the multivariate analysis, the variables in the age at first biopsy, ALT levels, presence of lymphoid aggregates and siderosis were the determinants of the best model for predicting the severity of the disease. The direct progression rate of hepatic structural lesions was significantly higher in untreated patients, intermediate in treated non-responders and lower in treated responders to antiviral therapy (non-treated vs responders, 0.22 ± 0.50 vs -0.15 ± 0.46, P = 0.0053). Immuno-expression of interferon receptor is not a relevant factor. CONCLUSION: The best predictors of the progression of fibrosis are age at the first liver biopsy, extent of ALT elevation, inflammation at liver histology and hepatic siderosis. Antiviral treatment is effective in preventing the progression of liver structural lesions in chronic hepatitis C patients. | Liliana SC Mendes Marcelo E Nita Suzane K Ono-Nita Evandro S Mello Luiz Caetano da Silva Venancio AF Alves Flair J Carrilho | 2008 | World Journal of Gastroenterology2008，14，16： | 0 |
您所在的机构暂未开通此部分的功能，请联系管理员