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408篇 您的检索式:作者名="MASLIAH E"
    题名 作者 年代 出处 被引量
1Immunoreactivity of CD45,a protein phosphotyrosine phosphatase,in Alzheimer' s disease 显示文摘Masliah E Mallory M Hansen L Alford M Albright T Terry R 1991Acta Neuropathol( Berl )1991,83,1:2
2Neurodegenerationin the central nervous system of apoE-deficient mice显示文摘Masliah E Mallory M Ge N 1995Exp Neurol1995,136,:1
3Profound increase in sensitivity to glutamatergic but not cholinergic agonistinduced seizures in transgenic mice with astrocyte production of IL-6显示文摘Samland H Huitron-Resendiz S Masliah E 2003J Neurosci Res2003,73,2:1
4Synaptophysin gene expression in human brain:a quantitative in sitn hybridization and immunocytochemical study显示文摘Eastwood SL Burnet PWJ Masliah E 1994Neuroscience1994,59,4:1
5The autophagy- related protein bcclin 1 shows reduced expression in early Alzheimer disease and regulates amyloid beta accumulation in mice显示文摘Piekford F Masliah E Britschgi M 2008J ClinInvest2008,118,6:1
6Physical basis of cognitive alterations in Alzheimer's disease:synapse loss is the major correlate of cognitive impairment显示文摘Terry RD Masliah E Salmon DP 1991Ann Neurol1991,30,:1
7Profound increase in sensitivity to glutamutergic but not cholinergic agonistinduced seizures in transgenic mice with astrocyte production of IL-6显示文摘Samland H Huitron-Resendiz S Masliah E 2003J Neurosci Res2003,73,2:1
8Dopaminergic loss andinclusion body formation in alpha-synuclein mice: implications for neurodegenerative disorders显示文摘Masliah E Rockenstein E Veinbergs I 2000Science2000,287,5456:1
9Immunohistochemical quantific-tion of the synapse-related protein synaptophysin in Alzheimer's disease显示文摘Masliah E Terry RD DeTeresa R 1989Neurosci Lett1989,103,2:1
10HSP70 reduces α-synuclein aggregation and toxicity显示文摘Klucken J Shin Y Masliah E 2004J Biol Chem2004,279,25:1
11Molecular mechanisms of neurodegeneration in Alzheimer's disease显示文摘Crews L Masliah E 2010Hum Mol Genet2010,19,1:1
12High-level neuronal expression of Aβ1-42 in wild-type human amyloid protein precursor transgenic mice: synaptotoxicity without plaque formation 显示文摘Mucke L Masliah E Yu GQ 2000J Neurosci2000,,:1
13Physical basis of cognitive altem-ations in Alzheimer's disease:synapse loss is the major correlate of cognitive impairment显示文摘Terry RD Masliah E Saimon DP 1991Ann Neurol1991,30,5:1
14Molecular cloning of cDNA encoding an unrecognized component of amyloid in Alzheimer disease 显示文摘 Fukushima H Masliah E 1993Proc Natl Acad Sci USA1993,90,11:1
15Profound increase in sensitivity to glutamatergic but not cholinergic agonistinduced seizures in transgenic mice with astrocyte production of IL-6显示文摘Samland H Huitron-Resendiz S Masliah E 2003J Neurosci Res2003,73,2:1
16β-Amyloid peptides enhance α-synuelein accumulation and neuronal deficits in atransgenic mouse model linking Alzheimer's disease and Parkinson's disease 显示文摘Masliah E Rockenstein E Veinbergs I 2001Neurobiology2001,98,12:1
17Physical basis of cognitive alterations in Alzheimer's disease:synapse loss is the major correlate of cognitive impairment显示文摘Terry R D Masliah E 1991Ann Neurol1991,30,:1
18Neuropathological alterations in Alzheimer dis- ease显示文摘Serrano-Pozo A Frosch MP Masliah E 2011Cold Spring Harb Perspect Med2011,1,00:1
19Alpha-synuclein in Lewy body disease and Alzheimer s disease显示文摘 Masliah E 1999Brain Pathol1999,9,6:1
20The precursor protein of non-A beta component of Alzheimer's disease amyloid is a presynaptic protein of the central nervous system显示文摘IwaiA Masliah E Yoshimoto M 1995Neuron1995,14,:1
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