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17篇 您的检索式:作者名="MARCOS Z D"
    题名 作者 年代 出处 被引量
1Effect of compost on rhizosphere microflora of the tomato and on the incidence of plant growth-promoting rhizobacteria显示文摘Brito D E Marcos A Alvare Z 1995Applied and Environmental Microbiology1995,61,1:1
2Milbemectin Resistance in Tetranychus urticae(Acari: Tetranychidag): Selection, Stabilityand Cross-resistance to Abamectin显示文摘ROBERTO L N MA'RIO E S MARCOS Z D 2010J Exp Appl Acarol2010,50,:1
3Evaluation of raw coals as fuels for direct carbon fuel cells显示文摘Li X Zhu Z H Marco R D 2010Journal of Power Sources2010,195,13:1
4Characterization of the RNA binding proteins forming complexes with a novel putative regulatory region in the 3-UTR of TNF-a mRNA显示文摘Hel Z Marco S D Radzioch D 1998Nucl Acids Res1998,26,11:1
5An in situ chronoamperometry/synchrotron radiation grazing incidence X-ray diffraction study of the electrochemical oxidation of pyrite in chloride media显示文摘Marco R D Bailey S Jiang Z T 2006Electrochemistry Communications2006,8,10:1
6Effect of compost on rhizospere microflora of the tomato and on the incidence of plant growth-promoting rhizobacteria显示文摘Brito D E Marcos A Alvare Z 1995Applied and Evironmental Microbiology1995,61,1:1
7Modification of coal as a fuel for the direct carbon fuel cell显示文摘Li X Zhu Z H Marco R D 2010Journal of Physical Chemistry A2010,114,11:1
8Factors that determine the performance of carbon fuels in the direct carbon fuel cell显示文摘Li X Zhu Z H Marco R D 2008Industrial & Engineering Chemistry Research2008,47,23:1
9Histologicalev aluation of 2 human immediately loaded implants inserted in the posterior mandible and retrieved after 6 months显示文摘 Giovanna P Giovanna Z 2003J Oral Implantol2003,5,2:1
10Fluorination of carbon fibers by halogen fluorides 显示文摘MARCOS Z DAN D HENRY S 1994Carbon1994,32,3:1
11Influence of postharvest chitosan treatment on enzymatic browning and antioxidant enzyme activity in sweet cherry fruit显示文摘Maria S P Donatella D Francesco M Luigi Z Marco S Milena P 2015Postharvest Biology and Technology2015,109,11:1
12Myelin-associated glycoprotein interacts with the Nogo 66 recepter to inhibit neurite outgrowth显示文摘MARCO D CAO Z TIMOTHY S 2002Neuron2002,35,2:1
13Characterization of the RNA binding proteins forming complexes with a novel putative regulatory region in the 3'UTR of TNF-α mRNA显示文摘 Marco S D Radzioch D 1998Nucleic Acids Research1998,26,28:1
14Bayesian networks for imputation显示文摘Marco D Z Mauro S 2004Journal of the Royal Statistical Society2004,167,2:1
15Myelin-associated glyeoprotein interacts with the Nogo-66 recepter to inhibit neurite outgrowth 显示文摘Marco D Cao Z Timothy S 2002Neuron2002,35,:1
16Hepatocellular carcinoma in nonalcoholic fatty liver disease:A growing challenge显示文摘Nonalcoholic fatty liver disease(NAFLD)is the most common cause of liver disease worldwide,and its prevalence increases continuously.As it predisposes to hepatocellular carcinoma both in the presence and in the absence of cirrhosis,it is not surprising that the incidence of NAFLD-related hepatocellular carcinoma would also rise.Some of the mechanisms involved in hepatocarcinogenesis are particular to individuals with fatty liver,and they help explain why liver cancer develops even in patients without cirrhosis.Genetic and immune-mediated mechanisms seem to play an important role in the development of hepatocellular carcinoma in this population.Currently,it is consensual that patients with NAFLD-related cirrhosis should be surveilled with ultrasonography every 6 mo(with or without alpha-fetoprotein),but it is known that they are less likely to follow this recommendation than individuals with other kinds of liver disease.Moreover,the performance of the methods of surveillance are lower in NAFLD than they are in other liver diseases.Furthermore,it is not clear which subgroups of patients without cirrhosis should undergo surveillance.Understanding the mechanisms of hepatocarcinogenesis in NAFLD could hopefully lead to the identification of biomarkers to be used in the surveillance for liver cancer in these individuals.By improving surveillance,tumors could be detected in earlier stages,amenable to curative treatments.Angelo Z Mattos Jose D Debes Renu Dhanasekaran Jihane N Benhammou Marco Arrese Andre Luiz V Patricio Amanda C Zilio Angelo A Mattos 2021World Journal of Hepatology2021,13,9:0
17Non-alcoholic fatty liver disease-related hepatocellular carcinoma:Is there a role for immunotherapy?显示文摘Hepatocellular carcinoma(HCC)is among the most common cancers and it is a major cause of cancer-related deaths.Non-alcoholic fatty liver disease(NAFLD)affects approximately one fourth of individuals worldwide and it is becoming one of the most important causes of HCC.The pathogenic mechanisms leading to NAFLD-related HCC are complex and not completely understood.However,metabolic,fibrogenic,oncogenic,inflammatory and immunological pathways seem to be involved.First-line therapy of advanced HCC has recently undergone major changes,since the combination of atezolizumab and bevacizumab was proven to increase survival when compared to sorafenib.Other immune-oncology drugs are also demonstrating promising results in patients with advanced HCC when compared to traditional systemic therapy.However,initial studies raised concerns that the advantages of immunotherapy might depend on the underlying liver disease,which seems to be particularly important in NAFLD-related HCC,as these tumors might not benefit from it.This article will review the mechanisms of NAFLD-related hepatocarcinogenesis,with an emphasis on its immune aspects,the efficacy of traditional systemic therapy for advanced NAFLD-related HCC,and the most recent data on the role of immunotherapy for this specific group of patients,showing that the management of this condition should be individualized and that a general recommendation cannot be made at this time.Ângelo Z Mattos Jose D Debes Arndt Vogel Marco Arrese Xavier Revelo Tales Henrique S Pase Muriel Manica Angelo A Mattos 2022World Journal of Gastroenterology2022,28,28:0
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