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66篇 您的检索式:作者名="MAHLKNECHT U"
    题名 作者 年代 出处 被引量
1Azacytidine causes complex DNA methylation responses in myeloid leukemia显示文摘Stresemann C Bokelmann I Mahlknecht U 2008Mol Cancer Ther2008,7,9:1
2Valproic acid and all-trans retinoic acid: meta-analysis of a palliative treatment regimen in AML and MDS patients显示文摘Bellos F Mahlknecht U 2008Onkologie2008,31,11:1
3The histone deacetylase inhibitor suberoylanilide hydroxamic acid down-regulates expression levels of Bcr-abl,c-Myc and HD AC3 in chronic myeloid leukemia cell lines显示文摘Xu Y Voelter-Mahlknecht S Mahlknecht U 2005Int J Mol Med2005,15,1:1
4SIRTUIN 1: regulating the regulator显示文摘Zschoernig B Mahlknecht U 2008Biochem Biophys Res Commun2008,376,2:1
5Role of histone deacetylase inhibitors in the treatment of cancer显示文摘Mei S Ho A D Mahlknecht U 2004Int J Oncol2004,25,6:1
6The histone deacetylase inhibitor suberoylanilide hydroxamic acid down-regulates expression levels of Bcr-abl,c-Myc and HDAC3 in chronic myeloid leukemia cell lines显示文摘Xu Y Voelter-Mahlknecht S Mahlknecht U 2005Int J Mol Med2005,15,1:1
7Histone deacetylase inhibitor treat-ment downregulates VLA-4 adhesion in hematopoietic stem cellsand acute myeloid leukemia blast cells显示文摘Mahlknecht U Schonbein C 2008Haematologica2008,93,3:1
8Valproic acid sensitizes chronic lymphocytic leukemia cells to apoptosis and restores the balance between pro-and antiapoptotic proteins显示文摘Bokelmann I Mahlknecht U 2008Mol Med2008,14,12:1
9The histone deace-tylase inhibitor suberoylanilide hydroxamic acid down-regulates expression levels of Bcr-abl,c-Myc and HDAC3 in chronic myeloid leukemia cell lines显示文摘Xu Y Voelter-Mahlknecht S Mahlknecht U 2005Int J Mol Med2005,15,1:1
10Valproic acid and all-trans retinoic acid:meta-analysis of palliative treatment regimen in AML and MDS patients显示文摘Bellos F Mahlknecht U 2008Onkologie2008,31,:1
11Cloning,chromosomal char-acterization and mapping of the NAD-dependent histone deacety-lases gene sirtuin 1 显示文摘Voelter-Mahlknecht S Mahlknecht U 2006Int J Mol Med2006,17,1:1
12Histone acetylation modifiers in the pathogensis of malignant disease显示文摘Mahlknecht U Hoelzer D 0,,08:1
13Valproic acid and all-trans retinoic acid:meta-analysis of a palliative treatment regimen in AML and MDS patients显示文摘Bellos F Mahlknecht U 2008Onkologie2008,31,11:1
14Stem cells and ageing The potential of stem cells to overcome age-related deteriorations of the body in regenerative medicine显示文摘Ho AD Wagner W Mahlknecht U 0,,:1
15Chromosomal organization and localization of the novel class IV human histone deacetylase 11 gene 显示文摘Voelter-Mahlknecht S Ho AD Mahlknecht U 2005Int J Mol Med2005,16,:1
16Azacytidine causes complex DNA methylation responses in myeloid leukemia 显示文摘Stresemann C Bokelmann I Mahlknecht U 2008Molecular Cancer Terapeutics2008,7,9:1
17The histone deacetylase inhibitor suheroylanilide hydroxamie acid down regulates expression levels of bcr-abl c-myc and HDAC3 in chronic myeloid leukemia cell lines显示文摘Xu Y Voelter-Mahlkneeht S Mahlknecht U 2005Int J Mol Med2005,15,1:1
18Histone acetylation modifiers in the pathogensis of malgnant disease 显示文摘MAHLKNECHT U HOELZER D 2000Mol Med2000,6,8:1
19Cloning and characterization of the murine histone deacetylase(HDAC3)显示文摘Mahlknecht U Hoelzer D Bucala R 1999Biochem Biophys Res Commun1999,263,2:1
20Histone acetylation modifiers in the pathogenesis of malignant disease显示文摘Mahlknecht U Hoelzer D 2000Mol Med2000,6,8:1
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