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56篇 您的检索式:作者名="MADDIKA S"
    题名 作者 年代 出处 被引量
1Cancer-specific toxicity of apoptin is independent of death receptors but involves the loss of mitochondrial membrane potential and the release of mitochondrial cell-death mediators by a Nur77-dependent pathway显示文摘Maddika S Booy EP Johar D 2005J Cell Sci2005,118,19:1
2WWP2 is an E3 ubiquitin ligase for PTEN显示文摘Maddika S Kavela S Rani N 2011Nat Cell Biol2011,13,6:1
3Cell survival, cell death and cell cycle pathways are interconnected: implications for cancer therapy 显示文摘Maddika S Ande SR Panigrahi S 2007Drug Resist Updat2007,10,12:1
4Cancer-selective therapy of the future:apoptin and its mechanism of action显示文摘Maddika S Mendoza F J Hauff K 2006Cancer Biol Ther2006,5,1:1
5Cell survival, cell death and cell cycle pathways are interconnected: implications for cancer therapy显示文摘Maddika S Ande SR Panigrahi S 2007Drug Resist Updat2007,10,:1
6Apoptin-induced cell death is modulated by Bcl-2 family members and is Apaf-1 dependent显示文摘Burek M Maddika S Burek C J 2006Oncogene2006,25,15:1
7Cancer-specific toxicity of apoptin is independent of death receptors but involves the loss of mitochondrial membrane potential and the release of mitochondrial cell-death mediators by a Nur77-dependent pathway显示文摘Maddika S Booy EP Johar D etal 2005Cell Sci2005,118,:1
8Cancer - speci? c toxicity of apoptin is independent of death receptors but involves the loss of mitochondrial membrane potential and the release of mi- toehondrial cell - deathmediators by a Nut77 - dependent pathway 显示文摘MADDIKA S BOOY E P JOHAR D 2005J Cell Sci2005,8,19:1
9Cancer selective therapy of the future:apoptin and its mechanism of action显示文摘MADDIKA S MENDOZA F HAUFF K 2006Cancer Biol Ther2006,5,1:1
10Monoclonal and bispecii?ic antibodies as novel therapeutics显示文摘Booy EP Johar 0 Maddika S 2006Arch Immunol Ther Exp2006,54,:1
11The ubiquitin pathway: an emer-ging drug target in cancer therapy显示文摘Ande SR Chen J Maddika S 2009Eur J Pharmacol?2009,625,3:1
12Mitochondrial physiology and toxicity; Importance for the immune system, Programmed cell death and cancer 显示文摘Pour-Jafari H Ghavami S Maddika S 2005Curr Med Chem-Anti - Inflammatory&Anti - Allergy2005,4,4:1
13Unscheduled Akt-triggered activation of cyclin-dependent kinase 2 as a key effector mechanism of apoptin's anticancer toxicity显示文摘Maddika S Panigrahi S Wiechec E 2009Mol Cell Biol2009,29,5:1
14Apoptin-induced cell death is modulated by Bcl-2 family members and is Apaf-1 dependent显示文摘Burek M Maddika S Burek CJ 2006Oncogene2006,25,15:1
15Cancer specific toxicity of apoptin is independent of death receptors but involves the loss of mitochondrial membrane potential and the release of mitochondrial cell death mediators by a Nur77-dependent pathway 显示文摘Maddika S Booy E P Johar D Gibson S B Ghavami S Los M 2005J Cell Sci2005,118,:1
16Unscheduled Akt- triggered activation of cyclin-dependent kinase 2 as a key effector mechanism of apoptin' s anticancer toxicity显示文摘Maddika S Panigrahi S Wiechec E 2009Mol CellBiol2009,29,5:1
17Cell survival,cell death and cell cycle pathways are interconnected:implications for cancer therapy显示文摘Maddika S Ande SR Panigrahi S 0,,1:1
18Apoptin-induced cell death is modulated by Bcl 2 family members and is Apaf- I dependent显示文摘Burek M Maddika S Burek C J 2006On-cogene2006,25,:1
19Apoptin-induced cell deathis modulated by Bcl-2 family members and is Apaf-1 dependent显示文摘Burek M Maddika S Burek CJ 2006Oncogene2006,25,15:1
20Cancer-specific toxicity of apoptin is independent of death receptors but involves the loss of mitochondrial membrane potential and the release of mitochondrial cell-death mediators by a Nur77-dependent pathway显示文摘MADDIKA S BOOY E P JOHAR D 2005J Cell Sci2005,118,19:1
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