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8篇 您的检索式:作者名="Lowenson JD"
    题名 作者 年代 出处 被引量
1Structural alterations inthe peptide backbone of beta-amyloid core protein may account forits deposition and stability in Alzheimer's disease 显示文摘Roher AE Lowenson JD Clarke S 1993J Biol Chem1993,268,5:1
2A new type of protein methylation activated by tyrphostin A25 and vanadate显示文摘Miranda TB Lowenson JD Clarke S 2004FEBS Lett2004,577,12:1
3B-Amyloid-(1~42)is a major component of cerebrovascular amyloid deposits: implica tions for the pathology of Alzheimer disease显示文摘Roher AE Lowenson JD Clarke S 1993Proc Xatl Acad Sci liSA1993,90,10:1
4Structural alterations in the peptidebackbone of beta-amyloid core protein may account for its de- position and stability in Alzheimer's disease显示文摘Roher AE Lowenson JD Clarke S 1993J Biol Chem1993,268,:1
5Beta-amyloid- (1-42)is a major component of cerebrovascular amyloid deposits: implications for the pathology of Alzheimer disease 显示文摘Roher AE Lowenson JD Clarke S 1993Proc Natl Acad Sci U S A1993,90,10:1
6Structural alterations in the peptide backbone of beta-amyloid core protein may account for its deposition and stability in Alzheimer's disease显示文摘Roher AE Lowenson JD Clarke S 1993J Biol Chem1993,268,5:1
7β-amyloid-(1-42) is a major component of cerebrovascular amyloid deposits:implications for the pathology of Alzheimer's disease显示文摘Roher AE Lowenson JD Clarke S 1993Proc Natl Acad Sci USA1993,90,:1
8Deficiency of a protein-repair enzyme results in the accumulatlon of altered proteins, retardation of growth, and fatal seizures in mice显示文摘Kim E Lowenson JD MacLaren DC 1997Proc Natl Acad Sci U S A1997,94,:1
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