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28篇 您的检索式:作者名="Lingrel JB"
    题名 作者 年代 出处 被引量
1Nal, K1-ATPase显示文摘Lingrel JB Kuntzweiler T 1994J Biol Chem1994,269,19:1
2Structure-function studies of the Na+, K+-ATPase显示文摘Lingrel JB Van Huysse J Brien W 1994Kidney Int1994,44,1:1
3Hypertension fromchronic central Sodium chloride in mice is mediated by theouabain-binding site on the Na, K-ATPase α2-isoform显示文摘Van Huysse JW Dostanic I Lingrel JB 2011Am JPhysiol Heart Circ Physiol2011,301,5:1
4Na,K-ATPase and the role of alpha isoforms in behavior显示文摘Lingrel JB Williams MT Vorhees CV 2007J Bioenerg Biomembr2007,39,56:1
5Molecular genetics of Na,K-ATPase显示文摘 Orlowski J Shull MM Price EM 1990Prog Nucl Acids Res and Mol Biol1990,38,1:1
6Na, K-ATPase and therole of alpha isoforms in behavior显示文摘Lingrel JB Williams MT Vorhees CV 2007J Bioenerg Biomembr2007,39,56:1
7Structure-function studies of the Na+ , K+-ATPase 显示文摘Lingrel JB Van Huysse J Brien W 1994Kidney Int Suppl1994,44,1:1
8Myeloid-specific Kriippel-like factor 2 inactivation increasesmacrophageand neutrophil adhesion and promotesatherosclerosis显示文摘Lingrel JB Pilcher-Roberts R Basford JE 2012Circ Res2012,110,10:1
9Na+/K+-ATPase a2- isoform preferentially modulates Ca2+ transients andsarcoplasmic reticulum Ca2+ release in cardiac myoeytes 显示文摘Despa S Lingrel JB Bers DM 2012Cardiovasc Res2012,95,4:1
10(+)/K(+) -ATPase α2-isoform preferentiallymodulates Ca2(+ ) transients and sarcoplasmic reticulumCa2(+ ) release in cardiac myocytes显示文摘Despa S Lingrel JB Bers DN 2012Cardiovasc Res2012,95,4:1
11Isoforms of the alpha subunit of Ha+ , K+ -ATPase and their significance 显示文摘Jewell EA Shamraj OI Lingrel JB 1992Acta Physiol Scand Suppl1992,607,:1
12Extracellular potassium dependence of the Na+-K+-ATP ase in cardiac myocytes: iso- form specificity and effect of phospholemman显示文摘Han F Tucker AL Lingrel JB 2009Am J Physiol Cell Physiol2009,297,:1
13Na,K-ATPase and the role ofαisoforms in behavior显示文摘Lingrel JB Williams MT Vorhees CV 2007J Bioenerg Biomembr2007,39,56:1
14Mice expressing ouabain-sensitive alpha1-Na,K-ATPase have increased susceptibility to pressure overload-induced cardiac hypertrophy显示文摘Wansapura AN Lasko VM Lingrel JB 2011American Journal of Physiology Heart and Circulatory Physiology2011,300,1:1
15Molecular genetics of Na,K- ATPase显示文摘Lingrel JB Orlowski J Shull MM 1990Prog Nucleic Acid Res Mol Biol1990,38,1:1
16Structure - function relationships in the Na, K - ATPase alpha subunit : site - directed mutagenesis of glutamine - 111 to arginine and asparagines - 122 to aspartic acid generates a ouabain - resistant enzyme 显示文摘Price EM Lingrel JB 1988Biochemistry1988,27,22:1
17Mye- loid-specific Kruppel-like factor 2 inactivation increases macrophage and neutrophil adhesion and promotes athero-sclerosis显示文摘Lingrel JB Pilcher-Roberts R Basford JE 2012Circ Res2012,0,10:1
18Na,K-ATPase: isoform structure, function, and expression 显示文摘Lingrel JB 1992J Bioenerg Biomembr1992,24,:1
19Na,K-ATPase:isoform structure,function,and expression显示文摘Lingrel JB 1992J Bioenerg Biomembr1992,24,3:1
20Isoforms of the alpha subunit of Na, K-ATPase and their significance 显示文摘Jewell EA Shamraj OI Lingrel JB 1992Acta Physiol Scand Suppl1992,607,:1
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