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13篇 您的检索式:作者名="Knapskog"
    题名 作者 年代 出处 被引量
1A Metric for Trusted Systems 显示文摘Josang A Knapskog S J 1998Global IT Security1998,,:1
2On stochastic modeling for integrated security and dependability evaluation显示文摘Sallhammar K Helvik B E Knapskog S J 2006Journal of Networks2006,1,5:1
3On periods of Edon-(2m,2k) family of stream cipher显示文摘GLIGOROSKI D MARKOVSKI S KNAPSKOG S J 2006SASC Stream Cipher Revisited2006,216,:1
4On stochastic modeling for integrated security and dependability evaluation显示文摘Sallhammar K Helvik B E Knapskog S J 0,,5:1
5Impairment based legislative limits for driving under the influence of non - alcohol drugs in Norway 显示文摘Vindenes V Jordbru D Knapskog A 2012Forensic Sci Int2012,219,13:1
6Fuzzy online risk assessment for distributed intrusion prediction and prevention systems显示文摘Kjetil Haslum Ajith Abraham and Svein Knapskog 2008Oomputer Modeling and Simulation2008,,:1
7A Metric for Trusted Systems显示文摘Josang A Knapskog S J 1998Wien : Austrian Computer Society1998,,4:1
8On stochastic modeling for integrated secruity and dependability evaluation显示文摘K Sallharnmar B E Helvik S J Knapskog 2006The Journal of Networks (S 1796-2056)2006,1,5:1
9The stream cipher edon80显示文摘Gligoroski D Markovski S Knapskog S J 2008Lecture Notes in Computer Science2008,4986,:1
10A metric for trusted systems显示文摘Josang A Knapskog SJ 1998Global IT1998,16,9:1
11On stochastic modeling for integrated security and dependability evaluation显示文摘SALLHAMMAR K HELVIK B E KNAPSKOG S J 2006Journal of Networks2006,1,5:1
12Multivariate quadratic trapdoor functions based on multivariate quadratic quasigroups显示文摘Gligoroski D Markovski S Knapskog S J 2008Math Proceedings of the American Conference on Applied Mathematics2008,,:1
13Associations of cerebrospinal fluid amyloidogenic nanoplaques with cytokines in Alzheimer’s disease显示文摘Background:The aggregation of amyloidβ(Aβ)is central in the pathogenesis of Alzheimer’s disease(AD).Recently it has been shown that specifically,larger,Thioflavin T-binding Aβaggregates are associated with increased neuroinflammation and cytokine release.This study was aimed to quantify fibrillary amyloid aggregates,so-called nanoplaques,and investigate their relationship with cytokines in the cerebrospinal fluid(CSF).Methods:CSF was collected from 111 patients assessed for cognitive complaints at the Oslo University Hospital Memory Clinic.The patients were grouped based on their amyloid status.The CSF nanoplaque concentration was quantified with the Thioflavin T-fluorescence correlation spectroscopy(ThT-FCS)assay.The levels of nine cytokines(eotaxin-1,granulocyte stimulating factor,interleukin[IL]-6,IL-7,IL-8,monocyte chemoattractant protein-1,gammainduced protein 10,macrophage inflammatory protein[MIP]-1α,and MIP-1β)were quantified with a magnetic bead-based multiplex assay and read on a Luminex IS 200 instrument.Results:There were 49 amyloid-negative and 62 amyloid-positive patients in the cohort;none of the cytokines differed significantly between the amyloid groups.The increased nanoplaque levels were associated with levels of MIP-1βbelow the lower limit of quantification,and with decreased levels of MIP-1αand IL-8.The associations remained significant when adjusted for age,sex,cognitive function,apolipoproteinε4 status and CSF core biomarker levels.Conclusion:The cytokine levels were not associated with amyloid status in this cohort.The nanoplaque levels were negatively associated with MIP-1β,MIP-1αand IL-8,which is in line with recent findings suggesting that the upregulation of some cytokine markers has a protective role and is negatively associated with AD progression.Mari Aksnes Hans Christian DAass Ann Tiiman Trine Holt Edwin Lars Terenius Nenad Bogdanovic Vladana Vukojevic Anne-Brita Knapskog 2021Translational Neurodegeneration2021,10,2:0
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