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| 1 | Comparison of imaging-based and pathological dimensions in pancreatic neuroendocrine tumors显示文摘AIM To establish the ability of magnetic resonance(MR) and computer tomography(CT) to predict pathologic dimensions of pancreatic neuroendocrine tumors(Pan NET) in a caseload of a tertiary referral center.METHODS Patients submitted to surgery for Pan NET at the Surgical Unit of the Pancreas Institute with at least 1 preoperative imaging examination(MR or CT scan) from January 2005 to December 2015 were included and data retrospectively collected. Exclusion criteria were: multifocal lesions, genetic syndromes, microadenomas or mixed tumors, metastatic disease and neoadjuvant therapy. Bland-Altman(BA) and Mountain-Plot(MP) statistics were used to compare size measured by each modality with the pathology size. Passing-Bablok(PB) regression analysis was used to check the agreement between MR and CT.RESULTS Our study population consisted of 292 patients. Seventy-nine(27.1%) were functioning Pan NET. The mean biases were 0.17 ± 7.99 mm, 1 ± 8.51 mm and 0.23 ± 9 mm, 1.2 ± 9.8 mm for MR and CT, considering the overall population and the subgroup of non-functioning-Pan NET, respectively. Limits of agreement(LOA) included the vast majority of observations, indicating a good agreement between imaging and pathology. The MP further confirmed this finding and showed that the two methods are unbiased with respect to each other. Considering ≤ 2 cm non-functioning-Pan NET, no statistical significance was found in the size estimation rate of MR and CT(P = 0.433). PBR analysis did not reveal significant differences between MR, CT and pathology.CONCLUSION MR and CT scan are accurate and interchangeable imaging techniques in predicting pathologic dimensions of Pan NET. | Salvatore Paiella Harmony Impellizzeri Elisabetta Zanolin Giovanni Marchegiani Marco Miotto Anna Malpaga Riccardo De Robertis Mirko D'Onofrio Borislav Rusev Paola Capelli Sara Cingarlini Giovanni Butturini Maria Vittoria Davì Antonio Amodio Claudio BassiAldo Scarpa Roberto Salvia Luca Landoni | 2017 | World Journal of Gastroenterology2017，23，17： | 4 |
| 2 | Gastroesophageal reflux and congenital gastrointestinal malformations显示文摘Although the outcome of newborns with surgical congenital diseases(e.g.,diaphragmatic hernia;esophageal atresia;omphalocele;gastroschisis) has improved rapidly with recent advances in perinatal intensive care and surgery,infant survivors often require intensive treatment after birth,have prolonged hospitalizations,and,after discharge,may have longterm sequelae including gastro-intestinal comorbidities,above all,gastroesophageal reflux(GER).This condition involves the involuntary retrograde passage of gastric contents into the esophagus,with or without regurgitation or vomiting.It is a well-recognized condition,typical of infants,with an incidence of 85%,which usually resolves after physiological maturation of the lower esophageal sphincter and lengthening of the intra-abdominal esophagus,in the first few months after birth.Although the exact cause of abnormal esophageal function in congenital defects is not clearly understood,it has been hypothesized that common(increased intra-abdominal pressure after closure of the abdominal defect) and/or specific(e.g.,motility disturbance of the upper gastrointestinal tract,damage of esophageal peristaltic pump) pathological mechanisms may play a role in the etiology of GER in patients with birth defects.Improvement of knowledge could positively impact the long-term prognosis of patients with surgical congenital diseases.The present manuscript provides a literature review focused on pathological and clinical characteristics of GER in patients who have undergone surgical treatment for congenital abdominal malformations. | Lucia Marseglia Sara Manti Gabriella D'Angelo Eloisa Gitto Carmelo Salpietro Antonio Centorrino Gianfranco Scalfari Giuseppe Santoro Pietro Impellizzeri Carmelo Romeo | 2015 | World Journal of Gastroenterology2015，21，28： | 3 |
| 3 | Targeting inflammation: new therapeutic approaches in chronic kidney disease (CKD) 显示文摘 | Impellizzeri D Esposito E Attley J | 2014 | Pharmacol Res2014，81，： | 1 |
| 4 | Molecular evidence for theinvolvement of PPAR-5 and PPAR-y in anti-inflammatory andneuroprotective activities of palmitoylethanolamide after spinal cordtrauma显示文摘 | Patemiti I Impellizzeri D Crupi R | 2013 | Neuroinflammation2013，10，： | 1 |
| 5 | Effects of Mitogen-Activated Pro- tein Kinase Signaling Pathway Inhibition on the Development of Cerulein-Induced Acute Pancreatitis in Mice 显示文摘 | Mazzon E Impellizzeri D Di Paola R | 2012 | Pan- creas2012，41，4： | 1 |
| 6 | A novel protective formulation of Palmitoylethanolamide in experimental model ofcontrast agent induced nephropathy显示文摘 | Cordaro M Impellizzeri D Bruschetta G | 2016 | Toxicol Lett2016，240，1： | 1 |
| 7 | Doco- sahexaenoic acid attenuates the early inflammatory re- sponse following spinal cord injury in mice: in-vivo and in-vitro studies显示文摘 | Paterniti I Impellizzeri D Di Paola R | 2014 | J Neuroinflammation2014，11，： | 1 |
| 8 | Docosahexaenoic acid attenuates the early inflammatory response following spinal cord injury in mice: in-vivo and in-vitro studies 显示文摘 | Paterniti I Impellizzeri D Di PR | 2014 | J Neuroinflammation2014，11，： | 1 |
| 9 | Combination therapy with melatonin and dexa-methasone in a mouse model of traumatic brain injury显示文摘 | Campolo M Ahmad A Crupi R Impellizzeri D Morabito R Esposito E Cuzzocrea S | | 0，，： | 1 |
| 10 | The NAMPT inhibitor FK866 reverts the damage in spinal cord injury 显示文摘 | ESPOSITO E IMPELLIZZERI D MAZZON E | 2012 | J2012，9，： | 1 |
| 11 | Administration of camosine in the treatment of acute spinal cord injury 显示文摘 | Di Paola R Impellizzeri D Salinaro AT | 2011 | Biochem Pharmacol2011，82，： | 1 |
| 12 | Effects of mitogen-activated protein kinase signaling pathway inhibition on the development of cerulein-induced acute pancreatitis in mice显示文摘 | Mazzon E Impellizzeri D Di paola R | 2012 | Pancreas2012，41，4： | 1 |
| 13 | Role of Toll like receptor 4 signaling pathway in the secondary damage induced by experimental spinal cord injury显示文摘 | Impellizzeri D Ahmad A Di Paola R | 2015 | Immunobiology2015，220，9： | 1 |
| 14 | Administration of palmitoylethanolamide (PEA) protects the neurovascular unit and reduces secondary injury after traumatic brain injury in mice显示文摘 | Abroad A Crupi R Impellizzeri D | 2012 | Brain Behav Immun2012，26，8： | 1 |
| 15 | The renal inju- ry and inflammation caused by ischemia-reperfusion are reduced by genetic inhibition of TNF-ctR1 : a comparison with infliximab treatment显示文摘 | Di Paola R Genovese T Impellizzeri D | 2013 | Eur J Pharmacol2013，700，13： | 1 |
| 16 | Molecular evidence for the involvement of PPAR: and PPAR-7 in anti-inflamma- tory and neuroprotective activities of plamitoylethanolamide after spinal cord trauma显示文摘 | Paterniti I Impellizzeri D Crupi R | 2013 | J Neuroinflammation2013，10，： | 1 |
| 17 | Fatigue analysis of cold- worked and interference fit fastener hole显示文摘 | RICH D L IMPELLIZZERI L F | 1977 | ASTM Special Technical Publication1977，637，： | 1 |
| 18 | The renal injury and inflammation caused by ischemia-reperfusion are reduced by genetic inhibition of TNF-αR1:a comparison with infliximab treatment显示文摘 | Di Paola R Genovese T Impellizzeri D | 2013 | Eur J Pharmacol2013，700，13： | 1 |
| 19 | Post-ischaemic thyroid hormone treatment in a rat model of acute stroke显示文摘 | Genovese T Impellizzeri D Ahmad A | 2013 | Brain Res2013，1513，： | 1 |
| 20 | Effects of palmi-toylethanolamide on intestinal injury and inflammation causedby ischemia-reperfusion in mice 显示文摘 | Di Paola R Impellizzeri D Torre A | 2012 | J Leukoc Biol2012，91，6： | 1 |
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