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	题名	作者	年代	出处	被引量
1	Angiogenic peptide hydrogels for treatment of traumatic brain injury显示文摘Traumatic brain injury(TBI)impacts over 3.17 million Americans.Management of hemorrhage and coagulation caused by vascular disruption after TBI is critical for the recovery of patients.Cerebrovascular pathologies play an important role in the underlying mechanisms of TBI.The objective of this study is to evaluate a novel regenerative medicine for the injured tissue after brain injury.We utilized a recently described synthetic growth factor with angiogenic potential to facilitate vascular growth in situ at the injury site.Previous work has shown how this injectable self-assembling peptide-based hydrogel(SAPH)creates a regenerative microenvironment for neovascularization at the injury site.Supramolecular assembly allows for thixotropy;the injectable drug delivery system provides sustained in vivo efficacy.In this study,a moderate blunt injury model was used to cause physical vascular damage and hemorrhage.The angiogenic SAPH was then applied directly on the injured rat brain.At day 7 post-TBI,significantly more blood vessels were observed than the sham and injury control group,as well as activation of VEGF-receptor 2,demonstrating the robust angiogenic response elicited by the angiogenic SAPH.Vascular markers von-Willebrand factor(vWF)andα-smooth muscle actin(α-SMA)showed a concomitant increase with blood vessel density in response to the angiogenic SAPH.Moreover,blood brain barrier integrity and blood coagulation were also examined as the parameters to indicate wound recovery post TBI.Neuronal rescue examination by NeuN and myelin basic protein staining showed that the angiogenic SAPH may provide and neuroprotective benefit in the long-term recovery.	Xiaotang Ma Agnieszka Agas Zain Siddiqui KaKyung Kim Patricia Iglesias-Montoro Jagathi Kalluru Vivek Kumar James Haorah	2020	Bioactive Materials2020，5，1：	3
2	Interactions of oxidative stress and neurovascular inflammation in the patho- genesis of traumatic brain injury显示文摘	Abdul-Muneer P M Chandra N Haorah J	2015	Mol Neurobiol2015，51，3：	1
3	Oxidative stress activates pro- tein tyrosine kinase and matrix metalloproteinases leading to blood- brain barrier dysfunction 显示文摘	Haorah J Ramirez SH Schall K	2007	J Neurochem2007，2，：	1
4	Blood–brain Barrier: Structural Components and Function Under Physiologic and Pathologic Conditions显示文摘	Yuri Persidsky Servio H. Ramirez James Haorah Georgette D. Kanmogne	2006	Journal of Neuroimmune Pharmacology2006，，3：	1
5	Rho-mediated regulation of tight junction during monocyte migration across blood-brain barrier in HIV-1 encephalitis (HIVE) 显示文摘	Persidsky Y Heilman D Haorah J Zelivyanskaya M Persidsky R Weber GA	2006	Blood2006，107，12：	1
6	Blood brain barrier: structural component and function under physiologic and pathologic conditions显示文摘	PERSIDSKY Y RAMIREZ SH HAORAH J	2006	Neuroimmune Pharmacol2006，1，：	1
7	Alcohol-induced oxidative stressin brain endothelial cells causes blood-brain barrier dysfunction显示文摘	Haorah J Knipe B Leibhart J	2005	JLeukoc Biol2005，78，：	1
8	Blood-brain barrier:structural components and function under physi-ologic and pathologic conditions显示文摘	Persidsky Y Ramirez SH Haorah J		0，，03：	1
9	Rho-mediated regulation oftight junctions during monocyte migration across the blood-brain bar-rier in HIV-1 encephalitis(HIVE) 显示文摘	Persidsky Y Heilman D Haorah J	2006	Blood2006，107，12：	1
10	Oxidative stress activates protein tyrosine kinase and matrix metalloproteinases leading to blood-brain barrier dysfunction 显示文摘	Haorah J Ramirez SH Schall K	2007	J Neurochem2007，，2：	1
11	Blood - brain barrier : structuralcomponents and function under physiologic and pathologic conditions 显示文摘	Persidsky Y Servio H Haorah R J	2006	J Neuroimmune Phamlacology2006，1，9：	1
12	Rho-mediated regula- tion of tight junctions during monocyte migration across the blood- brain barrier in HIV-1 encephalitis (HIVE) 显示文摘	Persidsky Y Heilman D Haorah J	2006	Blood2006，107，12：	1
13	Blood–brain Barrier: Structural Components and Function Under Physiologic and Pathologic Conditions显示文摘	Yuri Persidsky Servio H. Ramirez James Haorah Georgette D. Kanmogne	2006	Journal of Neuroimmune Pharmacology2006，，3：	1
14	Blood-brain barrier:structural components and function under physiologic and pathologic conditions显示文摘	Persidsky Y Ramirez SH Haorah J	2006	J Neuroimmune Pharmacol2006，1，3：	1
15	Rho-mediated regulation oftightjunctions during moneeyte migration across the blood-brain barrier in HIV-I encephalitis(HIVE)显示文摘	Heilman D Haorah J	2006	Blood2006，107，：	1
16	Blood brain barri- er:structural components and function under physiologic and pathologic conditions显示文摘	Persidsky Y Ramirez S H Haorah J	2006	J Neuroimmune Pharmaco[2006，1，22：	1
17	Blood-brain barrier; structural components and function under physiologic and pathologic conditions显示文摘	Persidsky Y Ramirez SH Haorah J	2006	J Neuroimmune Pharmacol2006，1，3：	1
18	Prevention of pancreatic cancer induction in hamsters by metformin 显示文摘	Schneider MB Matsuzaki H Haorah J	2001	Gastroenterology2001，120，5：	1
19	Nitrosation of glycine ethyl ester and ethyl diazoacetate to give the alkylating agent and mutagen ethyl chloro(hydroximino)acetate显示文摘	Zhou L Haorah J Chen S C	2004	Chem Res Toxicol2004，17，：	1
20	Activation of proteintyrosine kinases and matrix metalloproteinases causes blood -brainbarrier injury : novel mechanism for neurodegeneration associated withalcohol abuse显示文摘	Haorah J Schall K Ramirez SH	2008	Glia2008，56，1：	1

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