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268篇 您的检索式:作者名="Halestrap AP"
    题名 作者 年代 出处 被引量
1Elucidating the molecular mechanism of the permeability transition pore and its role in reperfusion injury of the heart显示文摘HALESTRAP AP KERR PM JAVADOV S 1998Biochim Biophys Acta1998,1366,12:1
2Mitochondrial non - specific pores remain closed during cardiac ischemia but open upon reperfusion 显示文摘Griffiths E J Halestrap AP 1995Biochem J1995,307,1:1
3Role of calcium ions in regulation of mammalian intramitochondria metabolism显示文摘Mccormack JG Halestrap AP Denton R 1990Physiol Rev1990,70,2:1
4The permeability transition pore complex:another view显示文摘Halestrap AP McStay GP Clarke SJ 2002Biochimie2002,84,2:1
5Transport of lactate and other monocarboxylates across mammalian plasma membranes显示文摘 Halestrap AP 1993Am J Physiol1993,264,:1
6Elucidating the molecular mechanism of the permeability transition pore and its role in reperfusion injufy of the heart 显示文摘Halestrap AP Kerr PM Javadov S 1998Biochem Biophys Acta1998,1366,12:1
7The adenine nucleotide translocase:a central component of the mitochondrial permeability transition pore and key player in cell death显示文摘Halestrap AP Brenner C 2003Curr med chem2003,10,16:1
8Sanglifehrin A acts as a potent inhibitor of the mitochondrial permeability transition and reperfusion injury of the heart by binding to cyclophilin - D at a different site from cyclosporin A显示文摘Clarke SJ MeStay GP Halestrap AP 2002J Biol Chem2002,277,34:1
9The mitochondrial phosphate car- tier interacts with eyclophilin D and may play a key role in the permeability lran- sition显示文摘Leung AW Varanyuwatana P Halestrap AP 2008J Biol Chem2008,283,26:1
10Sanglifehrin A acts as a potent inhibitor of the mitoehondrial permeability transition and reperfusion injury of the heart by binding to cyclophilin - D at a different site from cyclosporin A显示文摘Clarke SJ McStay GP Halestrap AP 2002Biol Chem2002,277,34:1
11The SLC16 gene family-from monocarboxylate transporters (MCTs) to aromatic amino acid transporters and beyond显示文摘Halestrap AP Meredith D 2004Pflügers Archiv2004,447,5:1
12Specific inhibition of pyruvate transport in rat liver mitochondria and human erythrocytes by n-cyano-dhydroxycinnamate显示文摘Halestrap AP Denton RM 1974Biochem J1974,138,2:1
13Mitochondrial non-specific pores remain closed during cardiac ischemia,but open upon repeffusion显示文摘Griffiths E Halestrap AP 0,,1:1
14The SLC16 gene family–structure,role and regulation in health and disease显示文摘Halestrap AP 2013Molecular aspects of medicine2013,34,2:1
15Mitochondrial permeability transition pore opening during myocardial reperfusion - a target for cardioprotection显示文摘Halestrap AP Clarke SJ Javadov SA 2004Cardiovasc Res2004,61,3:1
16The adenine nucleotide translocase:a central component of the mitochondrial permeability transition pore and key player in cell death显示文摘Halestrap AP Brennerb C 2003Curr Med Chem2003,10,16:1
17A pore way to die: the role of mitochondria in reperfusion injury and cardioprotection 显示文摘Halestrap AP 2010Biochem Soc Trans2010,38,4:1
18The plasma membrane lactate transporter MCT4, but not MCT1, is up-regulated by hypoxia through a HIF-lalpha-dependent mechanism显示文摘Ullah MS Davies AT Halestrap AP 2006J Biol Chem2006,281,14:1
19Mitochondria and cell death 显示文摘Halestrap AP Doran E Gillespie JP 2000Biochem Soc Trans2000,28,:1
20Mitochondrial non-specific pores remain closed during cardiac ischaemia but open upon reperfusion显示文摘Griffiths EJ Halestrap AP 1995Biochem J1995,307,:1
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