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| 1 | Curcumin and Apigenin – novel and promising therapeutics against chronic neuroinflammation in Alzheimer's disease显示文摘Alzheimer's disease is a progressive neurodegenerative disorder, characterized by deposition of amyloid beta, neurofibrillary tangles, astrogliosis and microgliosis, leading to neuronal dysfunction and loss in the brain. Current treatments for Alzheimer's disease primarily focus on enhancement of cholinergic transmission. However, these treatments are only symptomatic, and no disease-modifying drug is available for Alzheimer's disease patients. This review will provide an overview of the proven antioxidant, anti-inflammatory, anti-amyloidogenic, neuroprotective, and cognition-enhancing effects of curcumin and apigenin and discuss the potential of these compounds for Alzheimer's disease prevention and treatment. We suggest that these compounds might delay the onset of Alzheimer's disease or slow down its progression, and they should enter clinical trials as soon as possible. | Madhuri Venigalla Erika Gyengesi Gerald Münch | 2015 | Neural Regeneration Research2015，10，8： | 11 |
| 2 | Involvement of gap junctions in the manifestation and control of the duration of seizures in rats in vivo显示文摘 | Gajda Z Gyengesi E Hermesz E | 2003 | Epilepsia2003，44，12： | 1 |
| 3 | Oxidative stress in the hypothalamus:the importance of calcium signaling and mitochondrial ROS in body weight regulation显示文摘 | GYENGESI E PAXINOS G ANDREWS Z B | 2012 | Curr Neuropharmacol2012，10，4： | 1 |
| 4 | Involvement of gap junctions in the manifestation and control of the duration of seizures in rats in vivo显示文摘 | Cajda Z Gyengesi E Hermesz E | 2003 | Epilepsia2003，44，： | 1 |
| 5 | Corticosterone regulates synaptic input organization of POMC and NPY/Ag RP neurons in adult mice显示文摘 | Gyengesi E Liu ZW D'Agostino G | 2010 | Endocrinology2010，151，： | 1 |
| 6 | Corticosterone regulates synaptic input organization of POMC and NPY/Ag RP neurons in adult mice显示文摘 | Gyengesi E Liu ZW D’Agostino G | 2010 | Endocrinology2010，151，： | 1 |
| 7 | Involvement of gap junctions in the manifestation and control of the duration of seizures in rats in vivo显示文摘 | Gajda Z Gyengesi E Hermesz E | 2003 | Epilepsia2003，44，12： | 1 |
| 8 | Involvement of gap junctions in the manifestation and control of the duration of seizures in rats in vivo显示文摘 | Gajda Z Gyengesi E Hennesz E | 2003 | Epilepsia2003，44，12： | 1 |
| 9 | Involvement of gap junctions in the manifestation and control of the duration of seizures in rats in vivo 显示文摘 | Gajda Z Gyengesi E Hermesz E | 2003 | Epilepsia2003，44，12： | 1 |
| 10 | Distribution of secretagogin-containing neurons in the basal forebrain of mice, with special reference to the cholinergic corticopetal system显示文摘 | Gyengesi E Andrews ZB Paxinos G | 2013 | Brain Res Bull2013，94，5： | 1 |
| 11 | Involvement of gap junctions in the manifestation and control of the duration of seizures in rats in vivo显示文摘 | Gajda Z Gyengesi E Hermesz E | 2003 | Epilepsia2003，44，12： | 1 |
| 12 | Involvement of gap junctions in the manifestation and control of the duration of seizures in rats in vivo 显示文摘 | Gajda Z Gyengesi E Hermesz E | 2003 | Epilepsia2003，44，12： | 1 |
| 13 | Involvement of gap junctions in the manifestation and control of the duration of seizures in rats in vivo显示文摘 | GAJDA Z GYENGESI E HERMESZ E | 2003 | Epilepsia2003，44，： | 1 |
| 14 | Involvement of gap junctions in the manifestation and control of the duration of seizures in rats in vivo 显示文摘 | Gajda Z Gyengesi E Hermesz E | 2003 | Epilepsia2003，44，12： | 1 |
| 15 | Involvement of gap junctions in the manifestation and control of the duration of seizures in rats in vivo显示文摘 | Gajda Z Gyengesi E Hermesz E | | 0，，12： | 1 |
| 16 | The functional significance of gap junction channels in the epileptogenicity and seizure susceptibility of juvenile rats显示文摘 | Gajda Z Hermesz E Gyengesi E | 2006 | Epilepsia2006，47，6： | 1 |
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