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9篇 您的检索式：作者名="Graser RT"

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		题名	作者	年代	出处	被引量
	1	MHC class I-restricted T cells are required for all but the end stages of diabetes development in NOD mice and utilize a prevalent T cell receptor a-chain gene rearrangement显示文摘	DiLorenzo TP Graser RT Ono T	1998	Proc Natl Acad Sci1998，95，12：	1
	2	Genetic analysis of resistance to Type-1 Diabetes in ALR/Lt mice,a NOD-related strain with defenses against autoimmune-mediated diabetogenic stress 显示文摘	Mathews CF Graser RT Baglay RJ	2003	Immunogenetecs2003，55，7：	1
	3	Identification of a CD8 T cell that can independently mediate autoimmune diabetes development in the complete absence of CD4 T cell helper functions显示文摘	Dilorenson TP Wang F	2000	J Immunol2000，164，7：	1
	4	Identification of a CD8 T cell that can independently mediate autoimmune diabetes development in the complete absence of CD4 T cell helper functions显示文摘	Graser RT Dilorenzo TP Wang F	2000	J Immunol2000，164，7：	1
	5	Identification of a CD8 T cell that canindependently mediate autoimmune diabetes development in the completeabsence of CD4 T cell helper functions显示文摘	Graser RT Dilorenson TP Wang F	2000	J Immunol2000，164，7：	1
	6	Identification of a CD8 T cell that can independently mediate autoimmune diabetes devel- opment in the complete absence of CD4 T cell helper functions 显示文摘	Graser RT Dilorenson TP Wang F	2000	JImmunol2000，164，7：	1
	7	Identification of a CD8+T cell that can independently mediate autoimmune diabetes development in the complete absence of CD4+T cell helper functions 显示文摘	Graser RT DiLorenzo TP Wang F	2000	J Immunol2000，164，7：	1
	8	Major histocompatibilitycomplex class I-restricted T cells are required for all but the endstages of diabetes development in nonobese diabetic mice and usea prevalent T cell receptor alpha chain gene rearrangement显示文摘	DiLorenzo TP Graser RT Ono T	1998	Proc Natl Acad Sci USA1998，95，12：	1
	9	Identification of a CD8T cell that can independently mediate autoimmune diabetes development in the complete absence of CD4 T cell helper functions 显示文摘	Graser RT Dilorenson TP Wang F	2000	J Immunol2000，164，7：	1

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