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| 1 | Abeta oligomer-induced aberrations in synapse composition, shape, and den-sity provide a molecular basis for loss of connectivity inAlzheimer's disease 显示文摘 | Lacor PN Buniel MC Furlow PW | 2007 | J Neurosci2007，27，4： | 1 |
| 2 | A oligomer?induced aberrations in synapse composition, shape, and density provide a molecular basis for loss of connectivity in Alheimer' s disease显示文摘 | Lacor PN Buniel MC Furlow PW et sl | 2007 | Neuroscience2007，27，4： | 1 |
| 3 | Abeta oligomer- induced aberrations in synapse composition, shape,and density provide a molecular basis for loss of con- nectivity in Alzheimer's disease 显示文摘 | Laeor PN Buniel MC Furlow PW | 2007 | J Neurosci2007，27，4： | 1 |
| 4 | Abeta oligomer-induced aberrations in synapse composition, shape, and density provide a molecular basis for loss of connectivity in Alzheimer's disease 显示文摘 | Lacor PN Buniel MC Furlow PW | 2007 | J Neurosci2007，27，： | 1 |
| 5 | A gain-of-function mutation in the hif2a gene in familial erythrocytosis显示文摘 | Percy MJ Furlow PW Lucas GS | 2008 | N Engl J Med2008，358，2： | 1 |
| 6 | Abeta oligomer-induced aberrations in synapse composition,shape,and density provide a molecular basis for loss of connectivity in Alzheimer's disease显示文摘 | Lacor PN Buniel MC Furlow PW | 2007 | J Neurosci2007，27，： | 1 |
| 7 | Abeta oligomer-induced aberrations in synapse composition, shape, and density provide a molecular basis for loss of connectivity in Alzheimer' s disease 显示文摘 | Lacor PN Buniel MC Furlow PW | 2007 | J Neurosci2007，27，4： | 1 |
| 8 | A beta oligomer-induced aberrati ons in synapse composition, shape, and density provide a molecular basis for loss of connectivity in Alzheimer' s disease 显示文摘 | Lacor PN Bunie MC Furlow PW | 2007 | Ncurqsei2007，27，4： | 1 |
| 9 | A gain of function mutation in the hif2α gene in familial erythrocytosis 显示文摘 | Percy MJ Furlow PW Lucas GS | 2008 | N Engl J Med2008，358，2： | 1 |
| 10 | Abeta oligomer-induced aberrations in synapse composition, shape,and density provide a molecular basis for loss of connectivity in Alzheimer's disease 显示文摘 | Lacor PN Buniel MC Furlow PW | 2007 | Neuroscience2007，27，： | 1 |
| 11 | Aloligomer induced aberrations in synapse composition, shape, and density provide amoleeulm' basis for loss of connectivity in Alzhermer's disease显示文摘 | Lacor PN Buniel MC Furlow PW | 2007 | J Neurosci2007，27，4： | 1 |
| 12 | Abeta oligomer-induced aberrations in synapse composition,shape,and density provide a molecular basis for loss of connectivity in Alzheimer's disease显示文摘 | Lacor PN Buniel MC Furlow PW | | 0，，： | 1 |
| 13 | Abeta oligomer-induced aber- rations in synapse composition, shape, and density provide a molecular basis for loss of connectivity in Alzheimer's disease 显示文摘 | Lacor PN Buniel MC Furlow PW | 2007 | J Neurosci2007，27，： | 1 |
| 14 | Aβ oligomer-induced aberrations in synapse composition, shape, and density provide a molecular basis for loss of connectivity in Alzheimer's disease 显示文摘 | Lacor PN Buniel MC Furlow PW | 2007 | J Neurosci2007，27，4： | 1 |
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