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		题名	作者	年代	出处	被引量
	1	Interactions between histamine H3 and dopamine D2 receptors and the implications for striatal function 显示文摘	Ferrada C Ferre S Casado V	2008	Neuropharmacology2008，55，：	1
	2	Transthoracic focused rapid echocardiographic examination:realtimeevaluation of fluid status in critically ill trauma patients显示文摘	Ferrada P Murthi S Anand RJ	2011	J Trauma2011，70，1：	1
	3	Consequence analysis by means of characteristic curves to determinethe damage to buildings from the detonation of explosive substances as a function of TNT equivalence显示文摘	Fernando Di az Alonso Gonza′lez Enrique Ferrada S	2008	Journal of Loss Prevention2008，21，：	1
	4	Population study of the anchovy resource (Engraulis ringens Jenyns 1842 ) (Clupeiformes, Engraulidae) ,through DNA analysis 显示文摘	Ferrada S Hem6ndez K Montoya R et aI	2002	Gayana ( Concepci6n )2002，65，2：	1
	5	Overexpression of the cholesterol-binding protein MLN64 induces liver damage in the mouse显示文摘AIM: To examine the in vivo phenotype associated with hepatic metastatic lymph node 64 (MLN64) over-expression. METHODS: Recombinant-adenovirus-mediated MLN64 gene transfer was used to overexpress MLN64 in the livers of C57BL/6 mice. We measured the effects of MLN64 overexpression on hepatic cholesterol content, bile flow, biliary lipid secretion and apoptosis markers. For in vitro studies cultured CHO cells with transient MLN64 overexpression were utilized and apoptosis by TUNEL assay was measured. RESULTS: Livers from Ad.MLN64-infected mice exhibited early onset of liver damage and apoptosis. This response correlated with increases in liver cholesterol content and biliary bile acid concentration, and impaired bile flow. We investigated whether liver MLN64 expression could be modulated in a murine model of hepatic injury. We found increased hepatic MLN64 mRNA and protein levels in mice with chenodeoxycholic acid-induced liver damage. In addition, cultured CHO cells with transient MLN64 overexpression showed increased apoptosis. CONCLUSION: In summary, hepatic MLN64 over- expression induced damage and apoptosis in murinelivers and altered cholesterol metabolism. Further studies are required to elucidate the relevance of these fi ndings under physiologic and disease conditions.	Juan Enrique Tichauer María Gabriela Morales Ludwig Amigo Leopoldo Galdames Andrés Klein Verónica Quiones Carla Ferrada Alejandra Alvarez R Marie-Christine Rio Juan Francisco Miquel Attilio Rigotti Silvana Zanlungo	2007	World Journal of Gastroenterology2007，13，22：	0

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