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| 1 | Pre-ischemia electro-acupuncture potentiates the expression of Bcl-2 and transforming growth factor-beta 1 in rat brains显示文摘The expression of the anti-apoptotic molecules Bcl-2 and transforming growth factor-beta 1 is known to confer protective effects on the cerebral ischemia-reperfusion injury.The current study investigated the expression levels of Bcl-2 and transforming growth factor-beta 1 in response to multiple pre-ischemia electro-acupuncture at acupoints Zusanli(ST36)and Fengchi(GB20) stimulation.Rats were divided into five groups:uninjured,control,non-acupoint,GB20 and ST36. Rats in the non-acupoint,GB20 and ST36 groups received 30 minutes(3 times or 18 times)of electro-acupuncture stimulation before experimental cerebral ischemia was induced.Bcl-2 and transforming growth factor-beta 1 were found to be significantly increased in the ST36 groups with either 3 or 18 electro-acupuncture treatments(P<0.05).The production was higher with 18 electro-acupuncture treatments in the ST36 groups(P<0.05).In the GB20 groups,significant increase was only observed in transforming growth factor-beta 1 with 18 electro-acupuncture treatments(P<0.05).No significant elevation of the level of transforming growth factor-beta 1 was observed in the non-acupoint groups.However,the production of Bcl-2 increased with 18 treatments in the non-acupoint groups(P<0.05).The data suggest that multiple pre-ischemia electro-acupuncture at ST36 was effective in conferring neuroprotective effect on the brain by means of upregulation of Bcl-2 and transforming growth factor-beta 1 and the effect was increase with the number of treatment. | Ka Keung Yip Samuel CL Lo Kwok-fai So Dora MY Poon Mason CP Leung | 2012 | Neural Regeneration Research2012，7，24： | 3 |
| 2 | EDHF : spreading the in- fluence of the endothelium显示文摘 | Garland CJ Hiley CR Dora KA | 2011 | Br J Pharmaeol2011，164，3： | 1 |
| 3 | K+ is an endothelium-derived hyperpolarizing factor in rat arteries显示文摘 | Dora KA Gardener MJ | 1998 | Nature1998，396，6708： | 1 |
| 4 | K^+ is an endothelium-derived hyperpolarizing factor in rat arteries 显示文摘 | Edwards G Dora KA Gardener MJ et ai | 1998 | Nature1998，396，11： | 1 |
| 5 | Myoendothelial gap junction may provide the pathway for EDHF in mouse mesenteric artery 显示文摘 | Dora KA Sandow SL Gallagher NT el al | 2003 | J Vasc Res2003，40，： | 1 |
| 6 | K+ is an endothelium-derived hyperpolarizing factor in rat arteries显示文摘 | Edwards G Dora KA Gardener MJ Garland CJ Weston AH | 1998 | Nature1998，396，： | 1 |
| 7 | K+ is an endotheliun - derived hyperpolarizing factor in rat arteries显示文摘 | Dora KA Gardener MJ | 1998 | Nature1998，396，： | 1 |
| 8 | Myoendothelial gap junc- tions may provide the pathway for EDHF in mouse mesenteric artery 显示文摘 | Dora KA Sandow SL Gallagher NT | 2003 | J Vasc Res2003，40，5： | 1 |
| 9 | Evidence against C-type natriuretic peptide as an arterial 'EDHF' 显示文摘 | Garland CJ Dora KA | 2008 | Br J Pharmacol2008，153，1： | 1 |
| 10 | Role of heterocellular gap junction communication in endothelium-dependent smooth muscle hyperpolarization: Inhibition by a connexin-mimetic peptide显示文摘 | Martin PEM Chaytor AT Evans WH Garland CJ Griffith TM | 1999 | Biochem Biophys Res Commun1999，254，1： | 1 |
| 11 | K+ is an endothelium-derived hyperpolarizing factor in rat arteries显示文摘 | Dora KA Gardener MJ | 1998 | Nature1998，396，： | 1 |
| 12 | K+ is an EDHF in rat arteries显示文摘 | Dora KA Gardener MJ | 1998 | Nature (London)1998，396，6708： | 1 |
| 13 | Rapid endothelial cell-selective loading of connexin 40 antibody blocks endothelium-derived hyperpolarizing factor dilation in rat small mesenteric arteries显示文摘 | Dora KA Sandow SL | 2005 | Circ Res2005，97，： | 1 |
| 14 | K+ is an endothelium-derived hyperpolarizing factor in rat arteries显示文摘 | Edwards G Dora KA Gardener MJ | 1998 | Nature1998，396，： | 1 |
| 15 | K+ is an endothelium-derived hyperpolarizing factor in rat arteries显示文摘 | Edwards G Dora KA Gardener MJ | 1998 | Nature1998，396，： | 1 |
| 16 | Possible role for K《'+》 in endothelium-derived hyperpolarizing factor-linked dilatation in rat middle cerebral artery显示文摘 | McNeish A J Dora KA Garland CJ | 2005 | Stroke2005，，7： | 1 |
| 17 | Phospholemman磷酸化调节小鼠和人的血管张力、血压及高血压显示文摘平滑肌钠-钾-ATP酶(Na+-K+-ATPase, NKA)可调节血管张力和血压早已为人熟知,但其辅助蛋白phospholemman(PLM)的作用尚未明确。该研究的目的是验证如下假说:在体外,PLM磷酸化可调节血管张力;在体内和人体的实验模型中,这一机制对血管功能和血压的调节起着重要作用。 | 刘青(译) 练桂丽(摘/审校) Boguslavskyi A Tokar S Prysyazhna O Rudyk O Sanchez-Tatay D Lemmey HA Dora KA Garland CJ Warren HR Doney A Palmer CNA Caulfield MJ Vlachaki Walker J Howie J Fuller W Shattock MJ | 2021 | 中华高血压杂志2021，29，3： | 0 |
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