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45篇 您的检索式：作者名="DOU QP"

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	题名	作者	年代	出处	被引量
1	N-terminal cleavage of Bax by calpain generates a potent proapoptotic 18kD fragment that promotes Bcl-2-independent cytochrome G release and apoptotic cell death显示文摘	Gao G Dou QP	2001	J Cell Biochem2001，80，1：	1
2	Molecular mechanisms of green tea polyphenols 显示文摘	Dou QP	2009	Nutr Cancer2009，61，6：	1
3	Synthesis of phosphates and phosphates-acetates hybrids of green tea polyphenol ( - )- epigallocatechine-3-gallate (EGCG) and its G ring deoxy analogs as potential anticancer prodrugs 显示文摘	HUO C DOU QP CHAN TH	2011	Tetrahedron Letters2011，52，42：	1
4	Tea polyphenels and their roles in cancer prevention and chemotherapy显示文摘	Chen D Dou qP	2008	International Journal of Molecular Sciences2008，9，7：	1
5	Induction of a retinoblastoma phosphatase activity by anticancer drugs accompanies p53-independent G1 arrest and apoptosis 显示文摘	Dou QP An B Will PL	1995	Pro Natl Acad Sci USA1995，92，：	1
6	Targeting apoptosis pathway with natural terpenoids:implications for treatment of breast and prostate cancer显示文摘	Yang H Dou QP	2010	Curr Drug Targets2010，11，6：	1
7	A para-amino substituent on the D-ring of green tea polyphenol epigallocatechin-3-gallate as a novel proteasome inhibitor and cancer cell apoptosis inducer 显示文摘	Osanai K Landis-Piwowar KR Dou QP	2007	Bioorga&Med Chem2007，15，15：	1
8	Clinical development of novel proteasome inhibitors for cancer treatment 显示文摘	Yang H Zonder JA Dou QP	2009	Expert Opin Investig Drugs2009，18，7：	1
9	New applications of old metal -binding drugs in the treatment of human cancer 显示文摘	Schmitt SM Frezza M Dou QP	2013	Front Biosci ( Schol Ed)2013，4，：	1
10	Targeting apoptosis pathway with natural terpenoids: implications for treatment of breast and prostate cancer显示文摘	Yang H Dou QP	2010	Curr Drug Targets2010，11，6：	1
11	N - terminal cleavage of bax by calpain generates a potent proapoptotic 18- kD fragment that promotes bcl - 2 - independent cytochrome C release and apoptotic cell death显示文摘	Gao G Dou QP	2000	J Cell Biochem2000，80，1：	1
12	N-terminal cleavage of bax by calpain generates a potent proapoptotic 18-kDa fragment that promotes bcl-2-independ-ent cytochrome C release and apoptotic cell death显示文摘	Gao G Dou QP	2000	J Cell Biochem2000，80，1：	1
13	Regulatory processes affecting androgen receptor expression, stability, and function: potential targets to treat hormone-refractory prostate cancer显示文摘	REDDY GP BARRACK ER DOU QP	2006	J Cell Biochem2006，98，6：	1
14	N-terminal cleavage of bax by calpain gen- erates a potentproapoptotic 18-kDa fragment that promotes bcl-2-independent cytochrome C release and apoptotic cell death显示文摘	Gao G Dou QP	2000	J Cel lBiochem2000，80，1：	1
15	Cell cycle and drug sensitivity显示文摘	Kazi A Dou QP	2005	Methods Mol Med2005，1，：	1
16	Calmodulin-androgen receptor (AR) interaction: calcium-dependent, calpain-mediated breakdown of AR in LNCaP prostate cancer cells显示文摘	Pelley R P Chinnakannu K Murthy S Strickland F M Menon M Dou QP	2006	Cancer Res2006，66，11：	1
17	N-terminal cleavage of bax by calpain generates a potent proapoptotic 18-kDa fragment that promotes bcl-2-independent cytochrome C release and apoptotic cell death显示文摘	Gso G Dou QP	2000	J Cell Biochem2000，80，1：	1
18	Ester bond - containing tea polyphenols potently inhibit proteosome activity in vitro and in vivo显示文摘	Nam S Smith DM Dou QP	2001	J Biol Chem2001，276，13：	1
19	Clinical development of novel proteasome inhibitors for cancer treatment显示文摘	YANG H ZONDER JA DOU QP	2009	Expert Opin Investig Drugs2009，18，7：	1
20	N-terminal cleavage of Bax by calpain generates a potent proapoptotic 18-KD fragment that promotes Bcl-2 independent cytochrome C release and apoptotic cell death 显示文摘	Gao G Dou QP	2001	J Cell Biochem2001，80，1：	1

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