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| 1 | 卒中一级预防指南美国心脏协会/美国卒中协会对医疗专业人员的声明显示文摘这份更新版指南旨在为既往无卒中或短暂性脑缺血发作史的人群提供适时的综合性卒中预防循证建议,包括危险因素控制、头颈部动脉粥样硬化性疾病的干预方法以及预防血栓形成和血栓栓塞性卒中的抗栓治疗。此外,本指南还提供了针对遗传和药理学检测以及在其他特殊情况下(包括镰状细胞疾病和卵圆孔未闭)进行卒中预防的建议。 | James F. Meschia Cheryl Bushnell Bernadette Boden-Albala Lynne T. Braun Dawn M. Bravata Seemant Chaturvedi Mark A. Creager Robert H. Eckel Mitchell S.V. Elkind Myriam Fornage Larry B Goldstein Steven M. Greenberg Susanna E. Horvath Costantino Iadecola Edward C. Jauch Wesley S. Moore John A. Wilson 李迪 高一鹭 陈政弘 霍晓川 孙海欣 孙冬玲 茹小娟 江滨 | 2015 | 国际脑血管病杂志2015，23，6： | 4 |
| 2 | Hepatitis C eradication with sofosbuvir leads to significant metabolic changes显示文摘AIM To assess the effect of sofosbuvir(SOF) based regimens on glycemic and lipid control.METHODS This is a retrospective analysis of hepatitis C virus(HCV)-infected patients treated and cured with a SOF regimen [SOF/ribavirin/interferon, SOF/simeprevir, or SOF/ledipasvir(LDV) ± ribavirin] from January 2014 to March 2015. Patients with hemoglobin A1C(HbA1C) and lipid panels within six months before and six months after therapy were identified and included in our study. Due to the known hemolytic effect of ribavirin, HbA1C was obtained a minimum of three months post-treatment for the patients treated with a ribavirin regimen. Medical history, demographics, HCV genotype, pre-therapy RNA, and liver biopsies were included in our analysis. The patients who started a new medication or had an adjustment of baseline medical management for hyper-lipidemia or diabetes mellitus(DM) were excluded from our analysis.RESULTS Two hundred and thirty-four patients were reviewed, of which 60 patients met inclusion criteria. Sixty-three point three percent were male, 26.7% were Caucasian, 41.7% were African American and 91.7% were infected with hepatitis C genotype 1. Mean age was 60.6 ± 6.7 years. Thirty-nine patients had HbA1C checked before and after treatment, of which 22 had the diagnosis of DM type 2. HbA1C significantly decreased with treatment of HCV(pretreatment 6.66% ± 0.95% vs posttreatment 6.14% ± 0.65%, P < 0.005). Those treated with SOF/LDV had a lower HbA1C response than those treated with other regimens(0.26% ± 0.53% vs 0.71% ± 0.83%, P = 0.070). Fifty-two patients had pre- and post-treatment lipid panels; there was a significant increase in low-density lipoprotein(LDL) and total cholesterol(TC) after treatment(LDL: 99.5 ± 28.9 mg/dL vs 128.3 ± 34.9 mg/dL, P < 0.001; TC: 171.6 ± 32.5 mg/dL vs 199.7 ± 40.0 mg/dL, P < 0.001). Pretreatment body-mass index(BMI) did not differ from post-treatment BMI(P = 0.684). CONCLUSION Eradication of HCV with a SOF regimen resulted in a significant drop in HbA1C and an increase in LDL and TC post therapy. | Amilcar L Morales Zachary Junga Manish B Singla Maria Sjogren Dawn Torres | 2016 | World Journal of Hepatology2016，8，35： | 2 |
| 3 | Cardiac stem cells delivered intravascularly traverse the vessel barrier,regenerate infarcted myocardium,and improve cardiac function显示文摘 | STEIN AB URBANEK K et al | 2005 | Proc Natl Acad Sci U S A2005，102，10： | 1 |
| 4 | Bone marrow-derived mesenchymal stems cells and cardiac repair显示文摘 | Chugh AR Zuba-Surma EK Dawn B | 2009 | Minerva Car dioangiol2009，57，2： | 1 |
| 5 | IL-6 plays an obligatory role in late preconditioning via JAK-STAT signaling and upregulation of iNOS and COX-2显示文摘 | Dawn B Xuan YT Guo Y | 2004 | Cardiovasc Res2004，64，： | 1 |
| 6 | Cardiac stem cells delivered intravascularly traverse the vessel barriet, regenerate infarcted myocardium, and improve cardiac function 显示文摘 | Dawn B Stein AB Urbanek K | 2005 | Proc Natl Acad Sci USA2005，102，10： | 1 |
| 7 | IL-6 plays an obligatory role in late preconditioning via JAK-STAT signaling and upregulation of iNOS and COX-2显示文摘 | Xuan YT Guo Y | 2004 | Cardiovasc Res2004，64，1： | 1 |
| 8 | Cardiac stem cells delivered intravascularly traverse the vessel barrier,regenerate infracted myocardium,and improve cardiac function显示文摘 | DAWN B STEIN A B URBANEK K | 2005 | Proc Natl Acad Sci USA2005，102，10： | 1 |
| 9 | Bone marrow-derived pluripotent very small embryonic-like stem cells (VSELs) are mobilized after acute myocardial infarction 显示文摘 | Zuba-Surma EK Kucia M Dawn B | 2008 | J Mol Cell Cardiol2008，44，： | 1 |
| 10 | Role of the JAK-STAT pathway in protection against myocardial ischemia/reperfusion injury显示文摘 | Dawn B Xuan YT | 2003 | Trends Cardivasc Med2003，13，2： | 1 |
| 11 | Remote sensing image registra tion techniques: a survey显示文摘 | Dawn S Saxena V Sharma B | 2010 | Lecture Notes in Computer Sci ence2010，6134，： | 1 |
| 12 | Role of Src protein tyrosine kinases in late preconditioning against myocardial infarction显示文摘 | Dawn B Takano H Tang X L | 2002 | Am J Physiol Heart Circ Physiol2002，283，2： | 1 |
| 13 | Bone marrow-derived pluripotent very smal embryonic-like stem cells (VSELs) are mobilized after acute myocardial infarction显示文摘 | Zuba-Surma EK Kucia M Dawn B | | 0，，05： | 1 |
| 14 | Tricuspid regurgitation: pathophysiol- ogy and management 显示文摘 | Thapa R Dawn B Nath J | 2012 | Curr Cardiol Rep2012，14，2： | 1 |
| 15 | Two-dimensional and Doppler transesophageal echocardiographic delineation and flow characterization of anomalous coronary arteries in adults显示文摘 | Dawn B Talley JD Prince CR | 2003 | J Am Soc Echocardiogr2003，16，12： | 1 |
| 16 | Emerging role of the JAK-STAT pathway as a mechanism of protection against ischemia/reperfnsion injury显示文摘 | Bolli RR Dawn B Xuan Y | 2001 | J Mol Cell Cardiol2001，33，11： | 1 |
| 17 | Adult bone marrow-derived cells:regenerative potential,plasticity,and tissue commitment显示文摘 | Dawn B Bolli R | 2005 | Basic Res Cardiol2005，100，6： | 1 |
| 18 | Left atrial thrombus predicts transient ischemic attack in patients with atrial fibrillation 显示文摘 | Stoddard MF Singh P Dawn B | 2003 | Am Heart J2003，145，： | 1 |
| 19 | Delayed preconditioning-mimetic action of nitroglycerin in patients undergoing coronary angioplasty显示文摘 | Leesar MA Stoddard MF Dawn B Jasti VG Masden R Bolli R | 2001 | Circulation2001，2，： | 1 |
| 20 | Carbon monoxide induces a late preconditioning- mimetic cardioprotective and antiapoptotic milieu in the myocardium显示文摘 | Stein AB Bolli R Dawn B | 2012 | J Mol Cell Cardiol2012，52，1： | 1 |
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