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23篇 您的检索式：作者名="Calasanz"

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	题名	作者	年代	出处	被引量
1	Frequent gain of chromosome 19 in megakaryoblastic leukemias detected by comparative genomic hybridization显示文摘	Alvarez S MacGrogan D Calasanz MJ	2001	Genes Chromosomes Cancer2001，32，3：	1
2	Characterization of nonrandom chromosomal gains and losses in multiple myeloma by comparative genomic hybridization显示文摘	Cigudosa JC Rao PH Calasanz MJ	1998	Blood1998，91，8：	1
3	NALP1 is a transcriptional target for cAMP-response-element-binding protein (CREB) in myeloid leukaemia cells 显示文摘	Sanz C Calasanz M J Andreu E	2004	Biochem J2004，384，2：	1
4	Cytogenetic analysis of 280 patients with multiple myeloma and related dis- orders: primary breakpoints and clinical correlations 显示文摘	CALASANZ MJ CIGUDOSA JC ODERO MD	1997	Genes Chromosomes Cancer1997，18，2：	1
5	Analysis of genomic breakpoints in p 190 and p21 0 BCR-ABL indicate distinct mechanisms offormation显示文摘	ScoreJ Calasanz MJ Ottman 0	2010	Leukemia2010，24，10：	1
6	Analysis of genomic breakpoints in P190 and P210 BCR-ABL indicate distinct mechanisms of formation显示文摘	Score J Calasanz MJ Ottman O		0，，：	1
7	Analysis of genomic breakpoints in P190 and P210 BCR-ABL indicate distinct mechanisms of formation显示文摘	Score J Calasanz MJ Ottman O		0，，：	1
8	Characterization of nonrandom chromosomal gains and losses in multiple myeloma by comparative genomic hybridization显示文摘	Rao PH Calasanz MJ	1998	Blood1998，91，8：	1
9	Cytogenetic analysis of 280 patients with multiple myeloma and related disorders: primary breakpoints and clinical correlations 显示文摘	Calasanz MJ Cigudosa JC Odero MD	1997	Genes Chromosomes Cancer1997，18，2：	1
10	Cytogenetic analysis of 280 patients with multiple myeloma and related disorders: primary breakpoints and clinical correlations 显示文摘	Calasanz MJ Cigudosa JC Odero MD	1997	Genes Chromosomes Cancer1997，18，2：	1
11	Analysis of geneomic breakpoints in p190 and p210 BCR-ABL indicate distinct mecha- nisms of formation 显示文摘	Score J Calasanz MJ Ottman O	2010	Leukemia2010，24，10：	1
12	Cytogenetic analysis of 280 patients with multiple myeloma and related disorders: primary breakpoints and clinical correlations显示文摘	Calasanz M J Cigudosa J C Odero M D	1997	Genes Chromosom Cancer1997，18，：	1
13	Cytogenetic analysis of 280 patients with multiple myeloma and related disorders: primary breakpoints and clinical correlations 显示文摘	Calasanz MJ Cigudosa JC Odero MD	1997	Genes Chromosomes Cancer1997，18，2：	1
14	Frequent gain of chromosome 19 in megakaryoblastic leukemias detected by comparative genomic hybridization显示文摘	Alvarez S MacGrogan D Calasanz MJ	2001	G- enes Chromosomes Cancer2001，32，3：	1
15	P53 is frequently altered by methylation, mutation, and/or deletion in acute lymphoblastic leukemia显示文摘	Agirre X Novo FJ Calasanz M J	2003	Mol Carcinog2003，38，4：	1
16	Methylation of CpG dinucleotides and/or CCWGG motifs at the promoter of p53 correlates with decreased gene expression in a subset of acute lymphoblastic leukemia patients 显示文摘	Agirre X Vizmanos JL Calasanz MJ	2003	Onco- gene2003，22，7：	1
17	Cytogenetic analysis of 280 patients with multiple myeloma and related disorders: Primary breakpoints and clinical correlations显示文摘	Cigudosa JC Odero MD	1997	Genes Chromosomes Cancer1997，18，2：	1
18	Frequent gain of chromosome 19 in megakaryoblastic leukemias detected by comparative genomic hybridization显示文摘	MACGROGAN D CALASANZ M J	2001	Genes Chromosomes Cancer2001，32，：	1
19	Cytogenetics analysis of 280 patients with multiple myeloma and related disorder:primary breakpoints and clinical correlations显示文摘	Cigudosa J C Odero M D	1997	Genes Chromosom Cancer1997，18，2：	1
20	NALP1 is a transcriptional target for cAMP-response-elementbinding protein (CREB) in myeloid leukaemia cells 显示文摘	Sanz C Calasanz MJ Andreu E	2004	Biochem J2004，384，2：	1

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