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| 1 | Risk of cardiovascular,cardiac and arrhythmic complications in patients with non-alcoholic fatty liver disease显示文摘Non-alcoholic fatty liver disease(NAFLD)has emerged as a public health problem of epidemic proportions worldwide.Accumulating clinical and epidemiological evidence indicates that NAFLD is not only associated with liver-related morbidity and mortality but also with an increased risk of coronary heart disease(CHD),abnormalities of cardiac function and structure(e.g.,left ventricular dysfunction and hypertrophy,and heart failure),valvular heart disease(e.g.,aortic valve sclerosis)and arrhythmias(e.g.,atrial fibrillation).Experimental evidence suggests that NAFLD itself,especially in its more severe forms,exacerbates systemic/hepatic insulin resistance,causes atherogenic dyslipidemia,and releases a variety of pro-inflammatory,pro-coagulant and pro-fibrogenic mediators that may play important roles in the pathophysiology of cardiac and arrhythmic complications.Collectively,these findings suggest that patients with NAFLD may benefit from more intensive surveillance and early treatment interventions to decrease the risk for CHD and other cardiac/arrhythmic complications.The purpose of this clinical review is to summarize the rapidly expanding body of evidence that supports a strong association between NAFLD and cardiovascular,cardiac and arrhythmic complications,to briefly examine the putative biological mechanisms underlying this association,and to discuss some of the current treatment options that may influence both NAFLD and its related cardiac and arrhythmic complications. | Stefano Ballestri Amedeo Lonardo Stefano Bonapace Christopher D Byrne Paola Loria Giovanni Targher | 2014 | World Journal of Gastroenterology2014，20，7： | 55 |
| 2 | DNA methylation requires a DNMT1 ubiquitin interacting motif (UIM) and histone ubiquitination显示文摘 | Weihua Qin Patricia Wolf Nan Liu Stephanie Link Martha Smets Federica La Mastra Ignasi Forne Garwin Pichler David Horl Karin Fellinger Fabio Spada Ian Marc Bonapace Axel Imhof Hartmann Harz Heinrich Leonhardt | 2015 | Cell Research2015，25，8： | 5 |
| 3 | Edrophonium provocative testing during electrogastrography (EGG) : effects on dyspeptic symptoms and the EGG 显示文摘 | Bonapace ES Parkman HP Fisher RS | 1998 | Dig Dis Sei1998，43，： | 1 |
| 4 | Relation of elevated serumuric acid levels to incidence of atrial fibrillation in patients with type 2diabetes mellitus显示文摘 | Valbusa F Bertolini L Bonapace S | 2013 | Am J Cardiol2013，112，4： | 1 |
| 5 | Nonalcoholicfatty liver disease is associated with left ventricular dias-tolic dysfunction in patients with type 2 diabetes 显示文摘 | Bonapace S Perseghin G Molon G | 2012 | Dia-betes Care2012，35，2： | 1 |
| 6 | Association of nonalcoholic fatty liver disease with QTc interval in patients with type 2 diabetes显示文摘 | TARGHER G VALBUSA F BONAPACE S | 2014 | Nutr Metab Cardiovasc Dis2014，24，6： | 1 |
| 7 | Induction of autophagy-dependent necroptosis is required for childhood acute lymphoblastic leukemia cells to overcome glucocorticoid resistance显示文摘 | Bonapace L Bornhauser BC Schmitz M | 2010 | J Clin Invest2010，120，4： | 1 |
| 8 | Whole gut transit scintigraphy in the clinical evaluation of patients with upper and lower gastrointestinal symptoms显示文摘 | Bonapace E S Maurer A H Davidoff S | 2000 | The American journal of gas- troenterology2000，95，10： | 1 |
| 9 | Cessation of CCL2 inhibition accelerates breast cancer metastasis by promoting angiogenesis显示文摘 | BONAPACE L COISSIEUX MM WYCKOFF] | 2014 | Nature2014，515，7525： | 1 |
| 10 | Induction of autophagy-dependent necroptosis is required for childhood acute lymphoblastic leukemia cells to overcome glucocorticoid resistance显示文摘 | Bonapace L Bomhauser BC Schmitz M | 2010 | J Clin Invest2010，120，4： | 1 |
| 11 | Induction of autophagy-dependent necroptosis is required for childhood acute lymphoblastic leukemia cells to overcome glucocorticoid resistance显示文摘 | Bonapace L Bornhauser BC Schmitz M | 2010 | J Clin Invest2010，120，4： | 1 |
| 12 | Induction of autophagy-dependent necroptosis is required for childhood acute lymphoblastic leukemia cells to overcome glucocorticoid resistance显示文摘 | Bonapace L Bornhauser B C Schmitz M | 2010 | J Clin Invest2010，120，4： | 1 |
| 13 | Cytosolic carbonic anhydrase activity in chronic myeloid disorders with different clinical phenotype显示文摘 | Iuliano F Molica S | 2004 | Biochim Biophys Acta2004，1689，3： | 1 |
| 14 | Effects of ACE gene insertion/deletion polymorphism on response to spironolactone in patients with chronic heart failure 显示文摘 | Cicoira M Rossi A Bonapace S | 2004 | Am J Med2004，116，10： | 1 |
| 15 | Effects of a new class of NO-releasing NSAIDs platelets and isolated arteries 显示文摘 | Minuz P Lechi C Bonapace S | 1996 | Inflammopharmacology1996，4，： | 1 |
| 16 | Does fasting serum gastrin predict gastric acid suppression inpatient sonproton pum inhibitor 显示文摘 | Fisher RS Parkman HP | 2000 | Digestion2000，45，7： | 1 |
| 17 | Induction of autophagy-dependent necroptosis is required for childhood acute lymphoblastic leukemia cells to overcome glucocorticoid resistance 显示文摘 | Bonapace L Bornhauser BC Schmitz M | 2010 | J Clin Invest2010，120，4： | 1 |
| 18 | Differences in antimicrobial drug exposure in patients with various degrees of renal function based on recommendations from dosing references 显示文摘 | Bonapace CR White RL Frich LV | 2002 | Pharmacotherapy2002，22，9： | 1 |
| 19 | E-valuation of the Bonapace Method : a specific educationalintervention to reduce pain during childbirth显示文摘 | Bonapace Julie Chaillet Nils Gaumond Isabelle | 2013 | Journal ofpain research2013，6，11： | 1 |
| 20 | Comparison of methods of interpretation of checkerboard synergy testing 显示文摘 | Bonapace CR Bosso JA Friedrich LV | 2002 | Diagn Microbiol Infect Dis2002，44，4： | 1 |
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