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	题名	作者	年代	出处	被引量
1	Lactate influx through the endothelial cell monocarboxylate transporter MCT1 supports an NF-kappaB/IL-$ pathway that drives tumor angiogenesis 显示文摘	Vegran F Boidot R Michiels C	2011	Cancer Res2011，71，7：	1
2	Association of p53 gene alterations with the expression of antiapoptotic survivin splice variants in breast cancer 显示文摘	Vegran F Boidot R Oudin C	2007	Oncogene2007，26，2：	1
3	Tumour hypoxia determines the potential of combining mTOR and autophagy inhibitors to treat mammary tumours显示文摘	Seront E Boidot R Bouzin C	2013	Br J Cancer2013，109，10：	1
4	Transcriptional regulation of the survivin gene显示文摘	Boidot R Végran F Lizard-Nacol S	2014	Mol Biol Rep2014，41，1：	1
5	The role of telomeres in pre-dicting individual radiosensitivity of patients with cancer in the era ofpersonalized radiotherapy显示文摘	Mirjolet C Boidot R Saliques S	2015	Cancer Treat Rev2015，41，4：	1
6	Distinct expression of surviving splice variants in breast carcinomas显示文摘	Vegran F Boidot R Oudin C		0，，：	1
7	Distinct expression of Survivin splice variants in breast carcinomas显示文摘	Vegran F Boidot R Ouclin C	2005	Int J Oncol2005，27，4：	1
8	Overexpression of caspase-3s splice variant in locally advanced breast carcinoma is associated with poor response to neoadjuvant chemotherapy 显示文摘	V6gran F Boidot R Oudin C	2006	ClinCancerRes2006，12，19：	1
9	Lactate influx through the endothelial cell monocarboxylate transporter MCT1 supports an NF-κB/IL-8 pathway that drives tumor angiogenesis 显示文摘	VEGRN F BOIDOT R MICHIELS C	2011	Cancer Res2011，71，7：	1
10	Overexpression of caspase-3s splicevariant in locally advanced breast carcinoma is associated with poor response to neoadjuvant chemotherapy 显示文摘	Vegran F Boidot R Oudin C J	2006	Cancer Therapy : Clinical2006，12，19：	1
11	, The transcription factor IRFI dictates the IL-21-dependent anticancer functions of TH9 cells显示文摘	Vegran F Berger H Boidot R	2014	Nat Immunol2014，15，8：	1
12	Lactate influx through the en- dothelial cell monocarboxylate transporter MCT1 supports an NF-KB/IL-8 pathway that drives tumor angiogenesis 显示文摘	V6gran F Boidot R Michiels C	2011	Cancer Res2011，71，7：	1
13	A short caspase-3 isoform inhibits chemotherapy- induced apoptosis by blocking apoptosome assembly显示文摘	VEGRAN F BOIDOT R SOLARY E	2011	PLoS One2011，6，29：	1
14	Implication of alternative splice transcripts of caspase-3 and survivin in chemoresistance显示文摘	VEGRAN F BOIDOT R OUDIN C	2005	Bull Cancer2005，92，3：	1
15	Implication of alternativesplice transcripts of caspase-3 and survivin in chemoresistance显示文摘	Vegran F Boidot R Oudin C	2005	Bull Cancer2005，92，3：	1
16	Distinct expression of survivin splice variants in breast carcinomas显示文摘	Vegran F Boidot R Oudin C	2005	Int J Oncol2005，27，4：	1
17	Predictive value of survivin alternative transcript expression in locally advanced breast cancer patients treated with neoadjuvant chemotherapy显示文摘	Boidot R Vegran F Lizard Naeol S	2009	Int J Mol Med2009，23，2：	1
18	EGFR amplification induces sensitivity to antiEGFR therapy in pancreatic acinar cell carcinoma显示文摘Pancreatic acinar cell carcinoma(PACC) is a rare cancer. When the tumor is metastatic, few therapeutic options are available. Precision medicine using next-generation sequencing is defined by the administration of drugs based on the tumor genetic mutations. The usage of precision medicine for finding new therapeutic options for rare cancers is an emerging field. We have reported here the case of a patient bearing a multitreated metastatic PACC. This patient underwent somatic and constitutional exome analyses. The analyses revealed in the liver metastasis an amplification of the EGFR gene. Accordingly, the patient was treated with off-label usage of panitumumab. We observed rapid response with necrosis of the liver metastasis, while no efficacy was observed in the primary tumor. An exome analysis of the primary tumor revealed amplification of HER2 and MET with EGFR amplification. Such amplifications are known as a resistance mechanism to anti EGFR therapy. Our results suggest that exome analysis may be helpful to highlight targets in rare cancers, such as PACC. EGFR amplification in this pathology should be determined and could be used as a biomarker to propose anti EGFR therapy.	Corentin Richard Julie Niogret Romain Boidot Francois Ghiringhelli	2018	World Journal of Gastrointestinal Oncology2018，10，4：	1
19	Pt-modified Ni aluminides, MCrAlY-base multilayer coatings and TBC systemsfabricated by Spark Plasma Sintering for the protection of Ni-base superalloys 显示文摘	Monceau D Oquab D Estournes C Boidot M Selezneff S Thebault Y Cadoret Y		Surface and Coatings Technology0，204，67：	1
20	Association of p53 gene alterations with the expression of antiapoptotic survivin splice variants in breast cancer 显示文摘	Vegran F Boidot R Oudin C	2007	Oncogene2007，26，：	1

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