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| 1 | Mild wear of a low alloy steel at temperature up to 500 ℃显示文摘 | S S Athwal | 1983 | Tribology International1983，16，3： | 1 |
| 2 | Brain responses to changes in bladder volume and urge to void in healthy men显示文摘 | Athwal BS Berkley KJ Hussain l | 2001 | Brain2001，124，2： | 1 |
| 3 | Pathogenesis, investigation and dietary and medical management of gastroparesis 显示文摘 | Keld R Kinsey L Athwal V | 2011 | J Hum Nutr Diet2011，，24： | 1 |
| 4 | Pediatric infectious endophthalmitis:a review显示文摘 | Khan S Athwal L Zarbin M | 2014 | J Pediatr Ophthalmol Strabismus2014，51，3： | 1 |
| 5 | 14-3-3 proteins associate with the regulatory phosphorylation site of spinach leaf nitrate reductase in an isoform-specific manner and reduce de- phosphorylation of Ser-543 by endogenous protein phosphatases 显示文摘 | BACHMANN M Huber J L Athwal G S | 1996 | FEBS Letters1996，398，1： | 1 |
| 6 | Mild wear of a low alloy steel at temperatures up to500℃ 显示文摘 | Sullivan J L Athwal S S | 1983 | Tribologylntemational1983，16，3： | 1 |
| 7 | Biological significance of divalent metal ion binding to 14-3-3 proteins in relationship to nitrate reductase inactivation显示文摘 | ATHWAL G S HUBER J L HUBER S C | 1998 | Plant Cell Physiol1998，39，： | 1 |
| 8 | Mild wear of a low alloy steel at temperature up to 500 ℃显示文摘 | Sullivan J L Athwal S S | 1983 | Tribology International1983，16，3： | 1 |
| 9 | COVID-19 and hepatorenal syndrome显示文摘Coronavirus disease 2019(COVID-19)is a highly infectious disease which emerged into a global pandemic.Although it primarily causes respiratory symptoms for affected patients,COVID-19 was shown to have multi-organ manifestations.Elevated liver enzymes appear to be commonly observed during the course of COVID-19,and there have been numerous reports of liver injury secondary to COVID-19 infection.It has been established that patients with pre-existing chronic liver disease(CLD)are more likely to have poorer outcomes following COVID-19 infection compared to those without CLD.Co-morbidities such as diabetes,hypertension,obesity,cardiovascular and chronic kidney disease frequently co-exist in individuals living with CLD,and a substantial population may also live with some degree of frailty.The mechanisms of how COVID-19 induces liver injury have been postulated.Hepatorenal syndrome(HRS)is the occurrence of kidney dysfunction in patients with severe CLD/fulminant liver failure in the absence of another identifiable cause,and is usually a marker of severe decompensated liver disease.Select reports of HRS following acute COVID-19 infection have been presented,although the risk factors and pathophysiological mechanisms leading to HRS in COVID-19 infection or following COVID-19 treatment remain largely unestablished due to the relative lack and novelty of published data.Evidence discussing the management of HRS in highdependency care and intensive care contexts is only emerging.In this article,we provide an overview on the speculative pathophysiological me-chanisms of COVID-19 induced HRS and propose strategies for clinical diagnosis and management to optimize outcomes in this scenario. | Henry H L Wu Varinder S Athwal Philip A Kalra Rajkumar Chinnadurai | 2022 | World Journal of Gastroenterology2022，28，39： | 0 |
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