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418篇 您的检索式:作者名="Aljada"
    题名 作者 年代 出处 被引量
1Circulating mononuclear cells in the obese are in aprointlammatory state显示文摘Ghanim H Aljada A Hofmeyer D 2004Cireulation2004,110,12:1
2Inflammation : the link between insulin resistance,obesity and diabetes显示文摘Dandona P Aljada A Bandyopadhyay A 2004Trends Im- munol2004,25,1:1
3Metabolic syndrome:a comprehensive perspective based on interactions between obesity,diabetes,and inflammation显示文摘Dandona P Aljada A Chaudhuri A 0,,11:1
4Metabolic syndrome:a comprehensive perspective based on interactions between obesity,diabetes,and inflammation显示文摘Dandona P Aljada A Chaudhuri A 2005Circulation2005,111,11:1
5Nuclear factor-kappa B sup- pressive and inhibitor-kappa B stimulatory effects of tmglitazone in obese patients with type 2 diabetes : evidence of an anti-inflammato- ry action显示文摘Aljada A Garg R Ghanim H 2001J Clin Endocrinol Metab2001,86,7:1
6Upregulation of the tissue inhibitor of metalloproteinase-1 protein is associated with progression of human non-small-cell lung cancer显示文摘Aljada IS Ramnath N Donohue K 2004J Clin Oncol2004,22,16:1
7Insulin inhibits intranuclear nuclear factor KB and stimulates IКB in mononuclear cells in obese subjects:evidence for an anti-inflammation effect显示文摘Dandona P Aljada A Mohanty P 2001J Clin Endocrinol Metab2001,86,:1
8Effect of insulin on human aortic endothelial nitric oxide synthase显示文摘Aljada A Dandona P 2000Metabolism2000,49,:1
9Circulating rnononuclear cells in the obese are in a proinflammatory state 显示文摘Ghanim H Aljada A Hofmeyer D 2004Circulation2004,110,12:1
10Circulating mono-nuclear ceils in the obese are in a proinflammatory state显示文摘Ghanim H Aljada A Hofrneyer D 0,,12:1
11Glucose ingestion induces an increase in intranuclear nuclear factor kappaB,a fall in cellular inhibitor kappat3, and an increase in tumor necrosis factor alpha messenger RNA by mononuclear cells in healthy human subjects显示文摘Aljada A Friedman J Ghanim H 2006Metabolism2006,55,9:1
12Metabolic syndrome:a comprehensive perspective based on interactions between obesity, diabetes, and inflammation 显示文摘Dandona P Aljada A Chaudhuri A Circulation0,111,11:1
13Insulin inhibits NF-КB a nd MCP-1 expression in human aortic endothelial cells显示文摘Aljada A Ghanim H Saadeh R 2001J Clin Endocrinol Metab2001,86,:1
14Metabolic syndrome:a comprehensive perspective based on interactions between obesity,diabetes,and inflammation显示文摘Dandona P Aljada A Chaudhuri A 0,,11:1
15Insulin inhibits NFkappaB and MCP-1 expression in human aortic endothelial cells显示文摘 Ghanim H Saadeh R 2001J Clin Endocrinol Metab2001,86,1:1
16Insulin inhibits intranuclear nuclear factor κB and stimulates IκB in mononuclear cells in obese subjects:evidence for anti-inflammation effect显示文摘Dandona P Aljada A Mohanty P 2001J Clin Endocrinol Metab2001,86,7:1
17Suppression of nuclear factor-kappa B and stimulation of inhibitor kappa B by troglitazone:evidence for an anti-inflammatory effect and a potential antiatherosclerotic effect in the obese显示文摘Ghanim H Garg R Aljada A 2001J Clin Endocrinol Metab2001,86,3:1
18Circulating mononuclear cells in the obese are in a proinflammatory state显示文摘Ghanim H Aljada A Hofmeyer D 0,,12:1
19Insulin inhibits intranuclear factor kappaB and stimulates IkappaB in mononuclear cells in obese subjects: evidence for an anti-inflammatory effect? 显示文摘Dandona P Aljada A Mohanty P 2001J Clin Endocrinol Metab2001,86,7:1
20Increased plasma SOCS- 3concentration of macrophge migration inhibitory factor(MIF) and MIF mRNA in mononuclear cells in the obese and the suppressive action of mefform in显示文摘Dandona P Aljada A Ghanim H 2004J Clin Endocrinol Metab2004,89,11:1
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