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    题名 作者 年代 出处 被引量
1利用分子标记技术聚合3个稻瘟病基因改良金23B的稻瘟病抗性显示文摘以C101LAC和C101A51为稻瘟病抗性基因的供体亲本,金23B为受体亲本,通过杂交、复交及一次回交,在分离世代,利用分子标记辅助选择技术结合特异稻瘟病菌株接种鉴定和农艺性状筛选,获得6个导入Pi-1、Pi-2和Pi-33基因的金23B导入系,其中导入系W1对稻瘟病的抗病频率为96.7%,明显高于携带单个基因的C104LAC(Pi-1)、C101A51(Pi-2)和北京糯(Pi-33)。基因聚合后抗病频率提高,说明基因聚合是培育稻瘟病持久抗性的有效方法之一。陈红旗 陈宗祥 倪深 左示敏 潘学彪 朱旭东 2008中国水稻科学2008,22,1:97
2Physiological Macro-lesions Enhanced Resistance to Blast (Magnaporthe grisea) in Rice Near-isogenic Lines显示文摘Roll-leaf-1 (rl-1) and spot-leaf-1 (spl-1) were two near-isogenic lines, which were obtained after 3 to 4 backcrosses withearly season indica rice Zhefu 802 as recurrent parent. Henna macro-lesions, referred as physiological or morphological markers,began to appear on leaves at 4.5- to 6.0-leaf stage. The rice seedlings were inoculated at 3.5-, 5.0- and 7.0-leaf stages with highpathogenic races Zhong A1 and Zhong B1 of Magnaporthe grisea, respectively. The resistance of rl-1, spl-1 and Zhefu 802 againstblast was significantly different. The seedlings of Zhefu 802 at 3.5- to 7.0-leaf stage were susceptible to races Zhong A1 and ZhongB1 of M. grisea, whereas those of rl-1 and spl-1 at 3.5-, 5.0- and 7.0-leaf stages were susceptible, moderately resistant andresistant, respectively. These results suggested that the enhanced resistance of ri-1 and spl-1 related to the appearance of theirmorphological marker lesions. The experiment provided a basis for studying lesion mimic and hypersensitive response inassociation with disease resistance.HUANG Shi-wen Lu Ji-ying Luo Kun ZHANG Xiu-fu QIAN Qian 2005Rice science2005,12,2:1
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