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508篇 您的检索式:作者名="Finsterer"
    题名 作者 年代 出处 被引量
1Traumatic fatal cerebral hemorrhage in an old patient with a history of multiple sclerosis under dabigatran: a case report and review of the literature显示文摘直接抗凝剂药的一劣势是一粒解毒药的缺乏,它可以与创伤的大脑损害在病人变得相关。尽管有周期性的下降,有 atrial 纤维性颤动和中略的一个 77 年的老人收到了 dabigatran。由于 ground-level-fall,他与中线移动受不了 subarachnoidal 和 intraparenchymal 出血, subdural hematoma 和大脑浮肿。没有重获知觉,尽管有 osteoclastic 环锯术和 hematoma 撤退,他七天以后仍然保持无知觉、死。最可能,减少了由于增加的年龄,力量贡献了的 dabigatran 清理致命的功课。我们与 unexplained 下降在病人建议抑制抗凝剂治疗。如果抗凝剂治疗被认为必要,有他们为实验室监视和抗凝剂活动的颠倒的潜力的 vitamin-K-antagonists 应该被比较喜欢。Claudia Stollberger Andreas Ulram Adam Bastovansky Josef Finsterer 2015Journal of Geriatric Cardiology2015,12,1:4
2Beneficiality of combined levetiracetam, clonazepam for myoclonus in MERRF requires further confirmation显示文摘To the Editor: With interest, we read the article by Su et al[1] about the antiepileptic drug (AED) treatment of 17 patients with genetically confirmed myoclonic epilepsy with ragged-red fibers (MERRF) syndrome. It was found that monotherapy with either levetiracetam (LEV), clonazepam (CZP), valproic acid (VPA).Josef Finsterer 2019Chinese Medical Journal2019,,5:3
3左室肌小梁过多/致密化不全与卒中或栓塞Stllberger C. Finsterer J. 王永兴 2005世界核心医学期刊文摘(心脏病学分册)2005,0,8:3
4Genetic heterogeneity of noncompaction显示文摘To the Editor:With interest we read the article by Wu etal on a 53-year male with agenesia of the right lungand left-ventricular hypertrabeculation/noncompaction(LVHT).The paper raises the following concerns.LVHT is not exclusively congenital.In single cases it hasbeen shown to develop during life.How did previousechocardiograms and ECGs look like?Josef Finsterer Claudia Stllberger 2007Chinese Medical Journal2007,,18:2
5Cardiac involvement in primary myopathies 显示文摘FINSTERER J STOLLBERGER C 2000Cardiology JT-Cardiology2000,94,1:1
6Preoperative acute normovelmic hemodilution is an alternative to hypervolemic hemodilution-in case of proper use显示文摘Kreimeier U Finsterer U 1997Anesth Analg1997,85,:1
7Hereditary spastic paraplegias with autosomal dominant, recessive, X-linked, or maternal trait of inheritance 显示文摘Finsterer J L~scher W Quasthoff S 2012J Neurol Sci2012,318,12:1
8Left ventricular hypertrabeculation/noncompaction and association with additional cardiac abnormalities and neuromuscular disorders显示文摘Stollberger C Finsterer J Blazek G 2002Am J Cardiol2002,90,8:1
9Antipsychotic drugs and QT prolongation显示文摘Stollberger C Huber JO Finsterer J 2005Int Clin Psychopharmacol2005,20,5:1
10Left ventricular hypertrabecul ation/noncompaction显示文摘 Finsterer J Austria V 2004J Am Soc Echocardiogr2004,17,:1
11Left ventricular hypertrabeculation/ noncompaction 显示文摘Stollberger C Finsterer J 2004J Am Soc Echocardiogr2004,17,1:1
12Noncom- paction in a HIV - positive with cortical blindness as initial mani- festation of progressive muhlfocal leucencephalopathy 显示文摘Finsterer J Sttillberger C Coulibaly - Wirnmer M 2011Int J Cardio12011,149,1:1
13Genetic, pathogenetic, and phenotypic implications of the mitochondrial A3243G tRNA^Leu(UUR)mutation 显示文摘Finsterer J 2007Acta Neurologica Scandinavica2007,116,:1
14Cerebrospinal fluid neuron-specific enolase in non-selected patients显示文摘Finster J Exner M Rompold H 2004Scand J Clin Lab Invest2004,64,:1
15Ptosis:causes,presentation,and management显示文摘Finsterer J 2003Aesthetic Plast Surg2003,27,3:1
16Hypothyroid myopathy with unusually high serum creatine kinase values 显示文摘Finsterer J 1999Horm Res1999,52,4:1
17Complex mitochondriopathy associated with 4 mtDNA transitions显示文摘Finsterer J Bittner R Bodingbauer M 2000Eur Neurol2000,44,:1
18Hypoteyroid myopathy with unusuall highserum creak kinase Values显示文摘Finster J 1999Horm Res1999,52,4:1
19Transient left ventricular dys-function (tako - tsubo phenomenon) : Findings and potential pathophys-iological mechanisms显示文摘Stollberger C Finsterer J Schneider B 2006Can J Cardiol2006,22,12:1
20Influence of mitochondrion-toxic agents on the cardiovascular system 显示文摘Finsterer J Ohnsorge P 2013Regul Toxicol Pharmacol2013,67,3:1
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