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19篇 您的检索式:作者名="Ding, RG"
    题名 作者 年代 出处 被引量
1A Novel Method of the Modification of Ultrastable Y Zeolites With Citric Acid in Unbuffered System显示文摘The feasibility of Preparing high Si/Al ratio samples by treating USY zeolites withcitric acid solution in unbuffered system was extensively investigated. The effecd of treatingconditions was explored. The results show tha Si/Al ratios, crystallintites,contents of crystallinesilicon and thermal stabilities of the samples are grealy increased after moderate modification.Itmeans that it might be an effective method for the moderate modification of USYDing, RG Qian, L Song, LH Yan, ZF Wang, HP 1997Chinese Chemical Letters1997,8,10:2
2Perfluoroisobutylene-induced acute lung injury and mortality are heralded by neutrophil sequestration and accumulation 显示文摘Wang HM Ding RG Ruan JX 2001J Occu Health2001,43,6:2
3Perfluoroisobutylene-induced acute lung injury and mortality are heralded by neutrophil sequestration and accumulation显示文摘Wang HM Ding RG Ruan JX 0,,06:1
4Perfluoroisobutylene-in- duced acute lung injury and mortality are heralded by neutrophil sequestrAT1 on and accumulation 显示文摘Wang HM Ding RG Ruan JX 2001J Occup Health2001,43,6:1
5Radiosensitivity enhancement of human hepatocellular carcinoma cell line SMMC-7721 by sorafenib through the MEK/ERK signal pathway显示文摘DAI XF DING J ZHANG RG 2013International Journal of Radiation Biology:Covering the Physical Chemical Biological and Medical Effects of Ionizing and Non-ionizing Radiations2013,89,9:1
6Dynamic changes in O-GlcNAcylation regulate osteoclast differentiation and bone loss via nucleoporin 153显示文摘Bone mass is maintained by the balance between osteoclast-induced bone resorption and osteoblast-triggered bone formation.In inflammatory arthritis such as rheumatoid arthritis(RA),however,increased osteoclast differentiation and activity skew this balance resulting in progressive bone loss.O-GlcNAcylation is a posttranslational modification with attachment of a single O-linkedβ-D-N-acetylglucosamine(O-GlcNAc)residue to serine or threonine residues of target proteins.Although O-GlcNAcylation is one of the most common protein modifications,its role in bone homeostasis has not been systematically investigated.We demonstrate that dynamic changes in O-GlcNAcylation are required for osteoclastogenesis.Increased O-GlcNAcylation promotes osteoclast differentiation during the early stages,whereas its downregulation is required for osteoclast maturation.At the molecular level,O-GlcNAcylation affects several pathways including oxidative phosphorylation and cell-cell fusion.TNFαfosters the dynamic regulation of O-GlcNAcylation to promote osteoclastogenesis in inflammatory arthritis.Targeted pharmaceutical or genetic inhibition of O-GlcNAc transferase(OGT)or O-GlcNAcase(OGA)arrests osteoclast differentiation during early stages of differentiation and during later maturation,respectively,and ameliorates bone loss in experimental arthritis.Knockdown of NUP153,an O-GlcNAcylation target,has similar effects as OGT inhibition and inhibits osteoclastogenesis.These findings highlight an important role of O-GlcNAcylation in osteoclastogenesis and may offer the potential to therapeutically interfere with pathologic bone resorption.Yi-Nan Li Chih-Wei Chen Thuong Trinh-Minh Honglin Zhu Alexandru-Emil Matei Andrea-Hermina Györfi Frederic Kuwert Philipp Hubel Xiao Ding Cuong Tran Manh Xiaohan Xu Christoph Liebel Vladyslav Fedorchenko Ruifang Liang Kaiyue Huang Jens Pfannstiel Min-Chuan Huang Neng-Yu Lin Andreas Ramming Georg Schett Jörg H.W.Distler 2022Bone Research2022,10,4:1
7Issues of health and disease relating to coal use in southwestern China显示文摘Zheng BS Ding ZH Huang RG 1999Inter J Coal Geology1999,40,23:1
8Perfluoroisobutylene in- duced acute lung injury and mortality are heralded by neutrophil sequestration and accumulation显示文摘Wang HM Ding RG Ruan JX 2001J Occup Health2001,,43:1
9显示文摘Xing X Qiao SZ Ding RG 2006Carbon2006,44,2:1
10Cell injuries of the blood-air barrier in acute lung injury caused by perfluoroisobutylene exposure显示文摘Meng G Zhao J Wang HM Ding RG Zhang XC Huang CQ 2010J Occup Health2010,52,1:1
11Perfluoroisobutylene-induced acute lung injury and mortality are heralded by neutrophil sequestration and accumulation显示文摘Wang H Ding RG Yuan B 2001J Occup Health2001,43,6:1
12Perfluoroisobutylene induced acute lung injury and mortality are heralded by neutrophil sequeg- tration and accumulation 显示文摘Wang HM Ding RG Ruan JX 2001J Occu Health2001,43,6:1
13Perfluoroisobutylene- induced acute lung injury and mortality are heralded by neutrophil sequestration and accumulation 显示文摘Wang HM Ding RG Ruan JX 2001Occup Health2001,43,6:1
14Perfluoroisobutylene-induced acute lung injury and mortality are heralded by neutrophil sequestration and accumulation显示文摘Wang HM Ding RG Ruan JX Yuan BL Sun XH Zhang XC 2001J Occup Health2001,43,6:1
15Cel injuries of the blood-air barrier in acute lung injury caused by perfluoroisobutylene exposure显示文摘Meng G Zhao J Wang HM Ding RG Zhang XC Huang CQ 2010J Occup Health2010,52,1:1
16Perfluoroisobutylene-induced acute lung injury and mortality are heralded by neutrophil sequestration and accumulation 显示文摘Wang HM Ding RG Ruan JX 2001Occup Health2001,43,6:1
17Perfluoroisobutylene-induced acute lung injury and mortality are heralded by neutrophil sequestration and accumulation显示文摘Wang HM Ding RG Ruan JX 2001J Occup Health2001,43,6:1
18Perfluoroisobutylene-induced acute lung injury and mortality are heralded by neutrophil sequestration and accunmlation 显示文摘Wang HM Ding RG Ruan JX 2001J Occup Health2001,43,6:1
19Perfluoroisobutylene-induced acute lung injury and mortality are heralded by neutrophil sequestration and accumulation 显示文摘Wang HM Ding RG Ruan JX 2001Occup Health2001,43,6:1
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